The role of the dentist and the otolaryngologist in evaluating temporomandibular joint syndromes

Gelb, Harold; Calderone, Joseph P.; Gross, Seymour M.; Kantor, Martin E.

doi:10.1016/0022-3913(67)90173-4

Cited by 68 publications

(24 citation statements)

References 5 publications

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“…[6][7][8][9][10] In the temporomandibular joint -disordered population tinnitus prevalence is between 12-68%. [11][12][13][14] Although this is a common problem, it has not been studied in detail. The mechanism of tinnitus and factors influencing it have not yet been revealed.…”

mentioning

confidence: 99%

Effects of sleep bruxism related tinnitus on quality of life

Saltürk

Özçelik²,

Kumral³

et al. 2015

Kulak Burun Bogaz Ihtis Derg

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Objectives:This study aims to analyze the subjective and objective characteristics of tinnitus in sleep bruxism patients. Patients and Methods:The study included 57 patients (12 males; 45 females; mean age 33.89±12.50 years; range 19 to 55 years) with sleep bruxism and tinnitus (sleep bruxism group) and 24 patients (6 males, 18 females; mean age 43.75±16.19 years; range 21 to 58 years) only with tinnitus (control group). Sleep bruxism was diagnosed by the diagnostic criteria of American Academy of Sleep Medicine. Patients were performed pure tone audiometry to detect hearing thresholds at standard and high frequencies. Tinnitus frequency and loudness were assessed. Subjective aspects of tinnitus were identified by tinnitus handicap inventory. Results:The statistical analysis revealed that the sleep bruxism group had significantly lower hearing thresholds except 1000 Hz and 2000 Hz. Tinnitus frequency was between 3000 Hz and 18000 Hz in sleep bruxism group while it was between 6000 and 16000 Hz in control group with no statistically significant difference (p=0.362). Sleep bruxism group had significantly lower tinnitus loudness and tinnitus handicap inventory scores in comparison to control group (p=0.024 and p=0.000, respectively). Conclusion:Tinnitus caused by sleep bruxism and temporomandibular joint issues has higher frequency and lower loudness compared to patients with only tinnitus.Keywords: Bruxism; frequency; hearing; tinnitus handicap inventory; tinnitus. ÖZ Amaç:Bu yazıda uyku bruksizmi olan hastalarda tinnitusun öznel ve nesnel özellikleri incelendi. Hastalar ve Yöntemler:Çalışmaya uyku bruksizmi ve tinnitusu olan 57 hasta (12 erkek; 45 kadın; ort. yaş 33.89±12.50 yıl; dağılım 19-55 yıl) (uyku bruksizmi grubu) ile sadece tinnitusu olan 24 hasta (6 erkek, 18 kadın; ort. yaş 43.75±16.19 yıl; dağılım 21-58 yıl) (kontrol grubu) dahil edildi. Uyku bruksizmi tanısı Amerikan Uyku Tıbbı Akademisi diyagnostik kriterlerine göre konuldu. Hastalara saf ses odyometri uygulanarak standart ve yüksek frekanslardaki işitme eşikleri belirlendi. Tinnitus frekansı ve şiddeti değerlendirildi. Tinnitusun öznel yönleri tinnitus engel dökümü ile belirlendi. Bulgular: İstatistiksel değerlendirme uyku bruksizmi grubunda işitme eşiklerinin 1000 Hz ve 2000 Hz dışında anlamlı olarak daha düşük olduğunu gösterdi. Tinnitus frekansı uyku bruksizmi grubunda 3000 Hz ile 18000 Hz arasında iken kontrol grubunda 6000 Hz ile 16000 Hz arasındaydı ve farklılık istatistiksel olarak anlamlı değildi (p=0.362). Uyku bruksizmi grubunda tinnitus şiddeti ve tinnitus engel dökümü puanları kontrol grubuna göre anlamlı olarak daha düşüktü (sırasıyla p=0.024 ve p=0.000).Sonuç: Uyku bruksizmi ve temporomandibüler eklem sorunlarına bağlı tinnitus yüksek frekanslıdır ve sadece tinnitusu olan hastalara göre daha az şiddetlidir.

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mentioning

confidence: 99%

Effects of sleep bruxism related tinnitus on quality of life

Saltürk

Özçelik²,

Kumral³

et al. 2015

Kulak Burun Bogaz Ihtis Derg

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“…The mobility of the DML as it passes through the petrotympanic fissure is related to its structure (Couly and Hureau, 1976;Cesarani et al, 1991;Sato et al, 2008). An anatomical relationship between the auditory ossicles and the DML is one of the clinical causes of hearing loss and temporomandibular joint (TMJ) disorders (Gelb et al, 1967;Bernstein et al, 1969;Düker et al, 1972;Pinto, 1962;Ioannides and Hoogland, 1983;Rohlin et al, 1985;Loughner et al, 1989). A relationship between the DML, tympanic cavity and mandibular fossa was also unknown in spite of previous reports for the tympanic cavity using CT images (Petrus and Lo, 1997; Kurosaki et al, 1998; Lacout et al, 2005).…”

Section: Introductionmentioning

confidence: 94%

Anatomical study of the human discomallear ligament using cone beam computed tomography imaging and morphological observations

Arai

Sato

2011

Okajimas Folia Anat. Jpn.

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Summary: In the present study, the human discomallear ligament (DML) was observed in structures at both macroscopic and cone beam computed tomography levels. Assessments were made regarding the distribution of calcitonin-gene-related peptide (CGRP), protein gene-product (PGP) 9.5, and substance P (SP) of the DML based on immunohistochemical analyses of the anatomical properties of jaw movements using 27 Japanese human cadavers (mean, 79.3 ± 8.6 years; male, 74.9 ± 8.0; female, 82.8 ± 7.5). The DML of the anterior region was connected to the TMJ disc. The DML of the posterior region was attached to both the head and the anterior process of the malleus through the petrotympanic fissure, which formed a narrow channel. The structure of the petrotympanic fissure through the DML was attached to the malleus, and this structure was associated with the mobility of the malleus. In the anterior and posterior parts of the disc-associated connective tissue of the DML, CGRP-, PGP9.5-and SP-positive nerve fibers were located around numerous blood vessels, a condition which may be correlated with chronic pain syndrines disorders and the auditory system.

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“…Die in Tabelle Goodfriend [13] 168 (14) Hankey [15] 100 (1) Vero [50] 150 64 (42) 2 (2) Gelb et al [11] 742 263 (35) 311 (42) Myrhaug [27] 1986 444 (22) Posselt [32] 731 (ca. 4) Morgan [25] 90 60 (67) Carlsson et al [5] 350 Myrhaug [27], und später Arlen [1], versuchten, Tinnitus und andere Symptome (fluktuierende Hörfähigkeit, Vertigo, Wattegefühl im Ohr) bei negativem otologischem Befund über die gemeinsame Innervation der Kaumuskeln, des M. tensor veli palatini und des M. tensor tympani durch den N. trigeminus zu erklären.…”

Section: Map Und Ohrenschmerzenunclassified

Zum Zusammenhang zwischen Myoarthropathien des Kausystems und Ohrenbeschwerden (Otalgie, Tinnitus)

Türp

1998

HNO

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Speculations concerning a possible association between temporomandibular disorders (TMDs) and ear symptoms of otalgia and tinnitus have been made in the dental and medical literature for a long time. Current understanding of this association is reviewed. An analysis of relevant studies published between 1947 and 1996 shows that a relatively high percentage of TMD patients reported the presence of ear pain and noise. However, a cause-effect relationship has not been established to date. Theories that try to explain the concurrence of TMD symptoms and ear symptoms (particularly tinnitus) by a common underlying cause remain speculative. It has to be taken into consideration that due to their high prevalence, TMDs and ear symptoms in a given patients may be present by chance alone. A methodological weakness of most of the studies available is the fact that control groups were not considered. Owing to the lack of prospective, controlled, and randomized studies, statements regarding the effectiveness of certain dental measures for the treatment of otalgia and tinnitus in TMD patients are not well supported.

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The role of the dentist and the otolaryngologist in evaluating temporomandibular joint syndromes

Cited by 68 publications

References 5 publications

Effects of sleep bruxism related tinnitus on quality of life

Effects of sleep bruxism related tinnitus on quality of life

Anatomical study of the human discomallear ligament using cone beam computed tomography imaging and morphological observations

Zum Zusammenhang zwischen Myoarthropathien des Kausystems und Ohrenbeschwerden (Otalgie, Tinnitus)

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