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“…[21,112] In this regard it should be pointed isozyme population and restored the SR Ca 2+ -pump ATPase activout that etomoxir, the well known inhibitor of CPT-I, has been ity leading to a favorable effect on cardiac function in diabetic shown to prevent ischemic injury by increasing glucose utilization animals. J Card Fail 1998; 4: [67][68][69][70][71][72][73][74][75][76][77][78][79][80][81] inhibitor, methyl 2-tetraglycidate, has been reported to be useful in 2. [100] Other CPT-I inhibitors, namely 3sponse of the diabetic animals to treatment with etomoxir.…”
Section: Potential Use Of Cpt-i Inhibitors In Diabetes Mellitusmentioning
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“…[21,112] In this regard it should be pointed isozyme population and restored the SR Ca 2+ -pump ATPase activout that etomoxir, the well known inhibitor of CPT-I, has been ity leading to a favorable effect on cardiac function in diabetic shown to prevent ischemic injury by increasing glucose utilization animals. J Card Fail 1998; 4: [67][68][69][70][71][72][73][74][75][76][77][78][79][80][81] inhibitor, methyl 2-tetraglycidate, has been reported to be useful in 2. [100] Other CPT-I inhibitors, namely 3sponse of the diabetic animals to treatment with etomoxir.…”
Section: Potential Use Of Cpt-i Inhibitors In Diabetes Mellitusmentioning
“…CAC interacts with carnitine palmitoyltransferase 2 (CPT2), which is located on the inner side of the inner mitochondrial membrane and releases free l-carnitine and acyl-CoA, which can enter the β-oxidation pathway ( Figure 1). It needs to be added that carnitine acyl transferases are also present in the peroxisomes [11] and, since the oxidation of fatty acids in these organelles is not complete, it was suggested that shortened fatty acids in the form of acylcarnitines can be exported from peroxisomes for further oxidation in mitochondria [11]. Slc22a21 (Octn3) was argued to catalyze this reaction [12].…”
Section: Introductionmentioning
“…It is generally accepted that fatty acyl-CoA oxidase (FAO) is the rate-limiting enzyme for peroxisomal -oxidation. The fatty acid oxidation that takes place in the peroxisomes is independent of carnitine, but degraded long-chain fatty acyl groups are made available for mitochondrial -oxidation which is controlled by the carnitine palmitoyltransferase (CPT) system (see review by Ramsay, 1999). This process is probably regulated by CPT-I in both liver and muscle (McGarry and Brown, 1997).…”
Section: Introductionmentioning