The Pittsburgh Sleep Diary

Monk, Timothy H.; Reynolds, Charles F.; Kupfer, David J.; Buysse, Daniel J.; Coble, Patricia A.; Hayes, Amy J.; Machen, Mary Ann; Petrie, Susan R.; Ritenour, Angela

doi:10.1111/j.1365-2869.1994.tb00114.x

Cited by 461 publications

(306 citation statements)

References 16 publications

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“…It was an amended version of the Pittsburgh Sleep Diary (PghSD) which had been developed to measure perceptions of sleep timing and satisfaction (Monk et al 1994). Since there were several changes from the original PghSD, it was important to demonstrate that its validity had not been compromised.…”

Section: Methodsmentioning

confidence: 99%

Subjective sleep, depression and anxiety: inter‐relationships in a non‐clinical sample

Mayers

Grabau

Campbell

et al. 2009

Human Psychopharmacology

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Objectives: Previous research confirms the interdependent relationship between poor sleep and depression, but has often focused on objective measures of sleep and overlooked the importance of subjective factors.Insomnia may be maintained by anxiety and perceptions of poor sleep timing, and depression is associated with poor sleep satisfaction, regardless of perceived sleep timing. Methods:This study explored the contribution of current depression and anxiety to sleep perceptions.Participants (n=98) completed the Hospital Anxiety and Depression scale, and questionnaires were used to evaluate current and previous psychiatric illness, sleep disorders, and prescribed psychotropic medication.Results: A series of ANOVAs and regression analyses indicated that variance in sleep timing perceptions was more likely to be explained by symptoms of anxiety than depression; explained variance (adj. R 2 ) 25%, t = 2.361; p=.023. The analyses also showed that sleep satisfaction perceptions (adj. R 2 = 20%, t = 3.085; p=.004), and those relating to overall quality of life (adj. R 2 = 37%, t = -2.763; p=.013), was more likely to be explained by symptoms of depression. Conclusions:These findings support the observation that anxiety appears related to poorer sleep timing perceptions, while depression appears associated with poor sleep satisfaction. Further research is needed to explore the factors that might maintain poor sleep satisfaction in depression.

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Section: Methodsmentioning

confidence: 99%

Subjective sleep, depression and anxiety: inter‐relationships in a non‐clinical sample

Mayers

Grabau

Campbell

et al. 2009

Human Psychopharmacology

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“…Sleep duration was scored as the number of hours slept per night (from time the subject went to sleep until he/she got out of bed, minus minutes of sleep lost) and sleep efficiency as sleep duration divided by time in bed (time from lying down until getting out of bed) (cf. Monk et al, 1994). Both were averaged over the 14 days of data collection (or a minimum of 8 days for subjects missing interview data) to obtain average sleep duration and average sleep efficiency scores.…”

Section: Potential Mediating Variablesmentioning

confidence: 99%

Objective and subjective socioeconomic status and susceptibility to the common cold.

Cohen¹,

Alper²,

Doyle³

et al. 2008

Health Psychology

265

217

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Objective:We ask whether subjective socioeconomic status (SES) predicts who develops a common cold when exposed to a cold virus. Design: 193 healthy men and women ages 21-55 years were assessed for subjective (perceived rank) and objective SES, cognitive, affective and social dispositions, and health practices. Subsequently, they were exposed by nasal drops to a rhinovirus or influenza virus and monitored in quarantine for objective signs of illness and self-reported symptoms. Main Outcome Measures: Infection, signs and symptoms of the common cold, and clinical illness (infection and significant objective signs of illness). Results: Increased subjective SES was associated with decreased risk for developing a cold for both viruses. This association was independent of objective SES and of cognitive, affective and social disposition that might provide alternative spurious (third factor) explanations for the association. Poorer sleep among those with lesser subjective SES may partly mediate the association between subjective SES and colds. Conclusions: Increased Subjective SES is associated with less susceptibility to upper respiratory infection, and this association is independent of objective SES, suggesting the importance of perceived relative rank to health.

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“…Subjects kept the same sleep-wake schedule for each condition. During this time, they were asked to complete the French version of the 'Pittsburgh Sleep Diary' (Monk et al, 1994), and to report the amount as well as the different caffeinated products they had consumed (ie coffee, tea, chocolate, etc.). The mean number of milligrams of caffeine consumed per day was approximated for each subject according to the following criteria: 250 ml of coffee ¼ 100 mg caffeine; 250 ml of tea ¼ 50 mg of caffeine; 250 ml of cola ¼ 35 mg of caffeine; 10 g of chocolate ¼ 5 mg of caffeine.…”

Section: Research Protocolmentioning

confidence: 99%

Effects of Caffeine are more Marked on Daytime Recovery Sleep than on Nocturnal Sleep

Carrier

Fernandez‐Bolaños

Robillard

et al. 2006

Neuropsychopharmacol

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Caffeine is often used to counteract sleepiness generated by sleep deprivation, jet lag, and shift-work, and is consumed at different times of day. Caffeine also has effects on sleep. However, little is known about the interaction between sleep deprivation, circadian timing, and caffeine consumption on sleep. In this study, we compared the effects of caffeine on nocturnal sleep initiated at habitual circadian time and on daytime recovery sleep. Thirty-four moderate caffeine consumers participated in both caffeine (200 mg) and placebo (lactose) conditions in a double-blind crossover design. Seventeen subjects followed their habitual sleep-wake cycle and slept in the laboratory during the night (Night), while 17 subjects were sleep deprived for one night and recovery sleep started in the morning (DayRec). All subjects received a capsule of 100 mg of caffeine (or placebo) 3 h before bedtime, and the remaining dose 1 h before bedtime. Compared to placebo, caffeine lengthened sleep latency, increased stage 1, and reduced stage 2 and slow-wave sleep (SWS) in both groups. However, caffeine reduced sleep efficiency more strongly in the DayRec group, and decreased sleep duration and REM sleep only in that group. The stronger effects of caffeine on daytime recovery sleep compared to nocturnal sleep are probably the consequence of the combined influence of increasing circadian wake propensity drive and the dissipation of homeostatic sleep pressure. We propose that the reduction of SWS by caffeine during daytime sleep increases the impact of the circadian wake signal on sleep. These results have implications for individuals using caffeine during night time. Neuropsychopharmacology (2007) 32, 964-972.

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The Pittsburgh Sleep Diary

Cited by 461 publications

References 16 publications

Subjective sleep, depression and anxiety: inter‐relationships in a non‐clinical sample

Subjective sleep, depression and anxiety: inter‐relationships in a non‐clinical sample

Objective and subjective socioeconomic status and susceptibility to the common cold.

Effects of Caffeine are more Marked on Daytime Recovery Sleep than on Nocturnal Sleep

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