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“…The sensitivity of the HIS for the diagnosis of CFS in these studies was 91.4 % and the overall specificity was 85.1 % [11,12,25]. The high sensitivity and specificity of the FRAS and HIS for CFS improves on the moderate sensitivity and poor specificity of classical autonomic testing for CFS [2][3][4][5][6][7][8][9][10]. The fact that different methods, the FRAS and the HIS, demonstrated a distinct cardiovascular reactivity in CFS lends support to the existence of a distinct phenotype of the cardiovascular reactivity in CFS patients.…”
Section: Discussionmentioning
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“…The sensitivity of the HIS for the diagnosis of CFS in these studies was 91.4 % and the overall specificity was 85.1 % [11,12,25]. The high sensitivity and specificity of the FRAS and HIS for CFS improves on the moderate sensitivity and poor specificity of classical autonomic testing for CFS [2][3][4][5][6][7][8][9][10]. The fact that different methods, the FRAS and the HIS, demonstrated a distinct cardiovascular reactivity in CFS lends support to the existence of a distinct phenotype of the cardiovascular reactivity in CFS patients.…”
Section: Discussionmentioning
“…Evidence for this association comes from the overlap in symptoms between the two conditions, the common occurrence of neurally mediated hypotension during head-up tilt (HUTT) in patients with CFS, and the improvement of symptoms of CFS following therapy directed at neurally mediated hypotension [2][3][4]. The postural tachycardia syndrome, which has been attributed to a partial dysautonomia involving the vasculature of the legs, has also been described in CFS [5][6][7].…”
Section: Introductionmentioning
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“…Although no virus has been identified as the primary cause of CFS, the immune system seems to be fighting a virus in some patients, as evidenced by the RNase-L pathway. Evidence also points to neurological findings including hyperintense signals on MRI scans (Lange et al, 1998) and autonomic dysfunction (primarily neurally mediated hypotension; Schondorf and Freeman, 1999). In this review, we have only covered several of the subtypes that have been more extensively studied.…”
Section: Discussionmentioning
“…We hypothesize that an increased heart rate and lower stroke volume is caused by reduction of ventricular filling due to loss of venomotor tone or reduced blood volume, which in turn may result from a decreased level of physical activity [42]. Gravitational deconditioning due to decreased activity and prolonged bed rest is thought to play a role in the pathophysiological mechanism of OI in CFS patients [4,43]. Scores on mobility and ambulation in HUT-negative CFS patients were inversely correlated with the increase of blood pressure during tilt in HUT-negative CFS patients (r = 0.36, p = 0.033 and r = 0.36, p = 0.034 respectively).…”
Section: S Prevalence Of Oi In Cfs Patientsmentioning