2004
DOI: 10.1016/j.ijporl.2003.10.011
|View full text |Cite
|
Sign up to set email alerts
|

The effect of the season on otitis media with effusion resolution rates in the New York Metropolitan area

Help me understand this report

Search citation statements

Order By: Relevance

Paper Sections

Select...
2
1
1

Citation Types

1
15
1
3

Year Published

2006
2006
2023
2023

Publication Types

Select...
7
1

Relationship

0
8

Authors

Journals

citations
Cited by 28 publications
(20 citation statements)
references
References 9 publications
1
15
1
3
Order By: Relevance
“…The OAEs are believed to originate from active mechanical forces generated by the outer hair cells. The OAE can occur spontaneously or can be evoked by an acoustic stimulus [6].…”
Section: Discussionmentioning
confidence: 99%
“…The OAEs are believed to originate from active mechanical forces generated by the outer hair cells. The OAE can occur spontaneously or can be evoked by an acoustic stimulus [6].…”
Section: Discussionmentioning
confidence: 99%
“…OME is defined as inflammation of the middle ear space accompanied by fluid collection without signs or symptoms of acute infection such as otalgia, fever or a red, bulging tympanic membrane [2]. The mechanism of OME is still not clearly, although was determined inflammatory mediators such as prostaglandins (PG), leukotrienes (LT) and platelet-activating factors (PAF) have been identified in human OME, and in the experimentally induced OME of animals [3][4][5][6][7].…”
Section: Introductionmentioning
confidence: 99%
“…Along with these poor socio-economic status and unhygienic conditions and improper sanitation habits can further predispose to recurrent ear discharge. [8] [9] Predominant bacterial etiology of CSOM in our study was Pseudomonas aeruginosa seen in 40.65% of the cases followed by Staphylococcus aureus (14.63%) and Klebsiella aerogenes(11.78%). Pseudomonas, is the predominant cause of CSOM in tropical region does not usually inhabit the upper respiratory tract, its presence in the middle-ear cannot be ascribed to an invasion through ET and it should be considered as secondary invader gaining access to the middle-ear via defect in TM.…”
Section: Methodsmentioning
confidence: 65%