volume 66, issue 6, P3946-3949 1992
DOI: 10.1128/jvi.66.6.3946-3949.1992
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Abstract: The Bel-i protein of human foamy virus can activate transcription directed by the long terminal repeat (LTR) promoter of human immunodeficiency virus type 1 (HIV-1). The target sequence for Bel-i is shown to lie within the HIV-1 LTR U3 region but does not coincide with any previously described factor-binding site. Gene expression directed by an HIV-1 LTR lacking functional sites for the inducible cellular transcription factor NF-KB was activated over 100-fold by coexpression of Bel-1. These observations sugges…

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