2017
DOI: 10.1111/anec.12490
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Supraventricular tachycardia, pregnancy, and water: A new insight in lifesaving treatment of rhythm disorders

Abstract: Pregnancy may predispose to paroxysmal supraventricular tachycardia (SVT), in subjects with or without identifiable heart disease. Many physiological conditions such as autonomic nervous system changes, altered systemic hemodynamics, etc. can contribute to the onset of arrhythmias during pregnancy. Some cases reported the occurrence of arrhythmias in relation to systemic fluid variations. We report the case of a pregnant woman who experienced SVT due to fluid depletion, detected by bioimpedance vector analysis… Show more

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Cited by 8 publications
(8 citation statements)
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“…Beta-blockers, other than atenolol and calcium channel blockers are relatively safe and effective in managing SVT with minimal fetal and maternal risk. 12 Verapamil can get into the fetal circulation and cause fetal bradycardia in the first trimester and should be used with care. Adenosine remains the first line of treatment due to its ultrashort half-life, safety, and efficacy.…”
Section: Discussionmentioning
confidence: 99%
“…Beta-blockers, other than atenolol and calcium channel blockers are relatively safe and effective in managing SVT with minimal fetal and maternal risk. 12 Verapamil can get into the fetal circulation and cause fetal bradycardia in the first trimester and should be used with care. Adenosine remains the first line of treatment due to its ultrashort half-life, safety, and efficacy.…”
Section: Discussionmentioning
confidence: 99%
“…This medication has been successful in 84% of cases [ 23 ]. Beta-adrenergic blockers, such as propranolol and metoprolol, are anti-arrhythmia drugs that can be first-line for outpatient treatment and second-line for acute treatment [ 12 ]. Digoxin has been used in the first trimester of pregnancy and is relatively safe [ 23 ].…”
Section: Reviewmentioning
confidence: 99%
“…Hemodynamic changes in pregnancy result in an increased heart rate by at least 20% during the third trimester due to the fall in systemic vascular resistance [ 11 ]. This is displayed by the noticeably higher resting heart rate [ 12 ]. High amounts of estrogen have a significant effect on the cardiac tissue's excitability at the molecular level [ 13 ].…”
Section: Introductionmentioning
confidence: 99%
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“…Определенная роль отводится электролитному дисбалансу, в частности гипомагниемии, вследствие чего увеличивается активность синусового узла, что укорачивает время атриовентрикулярной передачи, снижает абсолютную рефрактерность и удлиняет рефрактерность относительную. В условиях внутриклеточного дефицита магния угнетается его способность ингибировать симпатическое влияние на сердце, возрастает потеря калия клеткой и увеличивается вариабельность длительности интервала QT [13,14].…”
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