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Cited by 57 publications
(43 citation statements)
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“…22 In addition, previous studies have shown that TNF-induced expression of cell adhesion molecules was repressed by statins in cultured ECs. [23][24][25] Moreover, we previously showed that KLF4 inhibited TNF-induced expression of VCAM1 through blocking the binding of NF-kB to the VCAM1 promoter in cultured ECs. 11 To clarify the molecular mechanisms whereby statins attenuated renal I/R injury in control mice, but not in Klf4 cKO mice, we examined whether KLF4 contributed to the suppressive effect of statins on TNF-induced VCAM1 expression in HUVECs.…”
Section: Klf4 Mediated the Suppressive Effect Of Statins On Tnf-inducmentioning
confidence: 99%
“…22 In addition, previous studies have shown that TNF-induced expression of cell adhesion molecules was repressed by statins in cultured ECs. [23][24][25] Moreover, we previously showed that KLF4 inhibited TNF-induced expression of VCAM1 through blocking the binding of NF-kB to the VCAM1 promoter in cultured ECs. 11 To clarify the molecular mechanisms whereby statins attenuated renal I/R injury in control mice, but not in Klf4 cKO mice, we examined whether KLF4 contributed to the suppressive effect of statins on TNF-induced VCAM1 expression in HUVECs.…”
Section: Klf4 Mediated the Suppressive Effect Of Statins On Tnf-inducmentioning
confidence: 99%
“…In contrast to studies where VCAM expression was monitored in lung homogenate or fixed lung sections, both of which do not provide spatial information (32,34,37), we chose quantum dot (QD) technology, wherein fluorescent QDs functionalized with antibodies toward adhesion molecules were used to detect VCAM expression in vitro and in vivo. Here we created QDs modified with endothelial-specific antibodies, either platelet endothelial cell adhesion molecule-1 (PECAM) as a stable endothelial marker, or VCAM as a proadhesive endothelial activation marker of lung inflammation in mice.…”
mentioning
confidence: 99%
“…It has been reported that pretreatment of human aortic ECs with atorvastatin reduces ROS liberation induced by tumor necrosis factor-α. 42 However, in our experimental model, cotreatment with atorvastatin did not show the same results and possibly denoting differences in experimental designs.…”
Section: Afu (Ros-dcf)mentioning
confidence: 53%