Staining of amyloid with cotton dyes

Yanagihara, Mamoru

doi:10.1001/archderm.120.9.1184

Cited by 27 publications

(5 citation statements)

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“…Unlike Congo red stain, these cotton dyes do not stain conditions like solar elastosis and colloid milium, and are therefore regarded as being more specific. 6 Furthermore, direct immu nofluorescence has shown immunoglobulin G, IgM and compliment C3 in the amyloid deposits. 4,7 Primary localized cutaneous amyloidosis is defined as localized amyloidosis of the skin without evidence of systemic involvement.…”

Section: Discussionmentioning

confidence: 99%

Cutaneous Amyloidosis: A Pilot Study at a Teaching Hospital in New Delhi

et al. 2017

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Section: Discussionmentioning

confidence: 99%

Cutaneous Amyloidosis: A Pilot Study at a Teaching Hospital in New Delhi

et al. 2017

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“…Another method to differentiate colloid from amyloid is that colloid does not react with Pagoda-red and other cotton dyes. [ 10 ] Immunohistochemically, colloid and amyloid are positive for amyloid-p protein, although this protein could be seen in normal elastic fibers and actinic elastosis. Amyloid frequently reacts with light-chain immunoglobulin, to which colloid shows no reaction.…”

Section: Discussionmentioning

confidence: 99%

Nodular colloid degeneration of the skin: Report of three cases with review and update

Ghanadan

Kamyab‐Hesari

Daneshpajouh

et al. 2014

Indian Dermatol Online J

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Nodular colloid degeneration (NCD) is a rare dermatological disorder and uncommon type of colloid milium. The degeneration may be related to sun exposure. In this report, three cases, all presenting as multiple plaques and nodules over the nose and the face are depicted. Histologically, these nodular masses were homogeneous, with eosinophilic-cleaved materials expanding the papillary dermis and extending into the deep dermis. Periodic acid Schiff (PAS), crystal violet, and methyl-violet stains highlight the colloid material. All the three cases were finally diagnosed as NCD. NCD could be considered in any case with facial nodules and a long history of sun exposure. We suggest the long-term sun exposure as an etiologic factor; thus sun protection would be the most preventive and available treatment.

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“…Although the blood test showed a moderate inflam matory syndrome with normal protein elec trophoresis, urine analysis revealed massive proteinuria and lambda light-chain BenceJones proteins. The Congo red and Dylon stainings [7] of sections from a periorbital skin biopsy revealed the presence of mas sive amyloid deposits in the dermis. The patient was emergency-hospitalized for as thenia.…”

Section: Case Reportmentioning

confidence: 95%

Primary Systemic Amyloidosis

Dezfoulian

Arrese

Fernández³

et al. 1996

Dermatology

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We report 2 cases of primary systemic amyloidosis. A monoclonal gammopathy was confirmed at the postmortem examination of the first patient. An extensive search for evidence of chronic infection, inflammation, neoplasms and paraproteinemia was conclusively negative in the other patient. The recognition of cutaneous signs of primary systemic amyloidosis is crucial to insure a rapid management aimed at postponing the fatal issue of the disease.

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Staining of amyloid with cotton dyes

Cited by 27 publications

References 0 publications

Cutaneous Amyloidosis: A Pilot Study at a Teaching Hospital in New Delhi

Cutaneous Amyloidosis: A Pilot Study at a Teaching Hospital in New Delhi

Nodular colloid degeneration of the skin: Report of three cases with review and update

Primary Systemic Amyloidosis

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