Semantic encoding deficits in frontal lobe disease and amnesia

Comparison of figural intrusion errors in three amnesic subgroups

“…Thus, it must have been the two deficits combined that caused the increased number of intrusion errors. These findings are consistent with other reports indicating that in both Korsakoff patients (Freedman & Cermak, 1986) and ACoA patients (DeLuca, 1993), this same combination of deficits produced the greatest impairment.…”

Comparison of figural intrusion errors in three amnesic subgroups

“…Thus, it must have been the two deficits combined that caused the increased number of intrusion errors. These findings are consistent with other reports indicating that in both Korsakoff patients (Freedman & Cermak, 1986) and ACoA patients (DeLuca, 1993), this same combination of deficits produced the greatest impairment.…”

Cognitive impairment following frontal lobe damage and its relevance to human amnesia.

“…with frontal lobe lesions tested in the present study did exhibit release from proactive interference. One possibility is that the impairment observed in KorsakofTs syndrome is the result of combined damage to frontal and diencephalic regions, as suggested by Freedman and Cermak (1986). Taken together, the findings from our study suggest that certain features of KorsakofTs syndrome may be attributed to frontal lobe dysfunction.…”

Neuropsychological correlates of short-term memory distractor tasks among patients with surgical repair of anterior communicating artery aneurysms.

“…ACoA patients did not differ significantly from controls on any of the dependent measures stemming from the release-from-PI task (see Table 3). Furthermore, when analyses identical to those performed by Freedman and Cermak (1986) were used, no differences in degree of release (Trial 5 − Trial 4) were found between ACoAs and controls on either the first block (first shift condition) or the sum across blocks. Although ACoA patients and controls did not differ in their performance on the 0-s delay trial of the VCT task, they did differ significantly in their performance on the other delay trials (3 s, 9 s, and 18 s combined), t (42) = 2.09, p < .05…”

Comparison of figural intrusion errors in three amnesic subgroups

“…Thus, it must have been the two deficits combined that caused the increased number of intrusion errors. These findings are consistent with other reports indicating that in both Korsakoff patients (Freedman & Cermak, 1986) and ACoA patients (DeLuca, 1993), this same combination of deficits produced the greatest impairment.…”

Cognitive impairment following frontal lobe damage and its relevance to human amnesia.

“…with frontal lobe lesions tested in the present study did exhibit release from proactive interference. One possibility is that the impairment observed in KorsakofTs syndrome is the result of combined damage to frontal and diencephalic regions, as suggested by Freedman and Cermak (1986). Taken together, the findings from our study suggest that certain features of KorsakofTs syndrome may be attributed to frontal lobe dysfunction.…”

Neuropsychological correlates of short-term memory distractor tasks among patients with surgical repair of anterior communicating artery aneurysms.

“…ACoA patients did not differ significantly from controls on any of the dependent measures stemming from the release-from-PI task (see Table 3). Furthermore, when analyses identical to those performed by Freedman and Cermak (1986) were used, no differences in degree of release (Trial 5 − Trial 4) were found between ACoAs and controls on either the first block (first shift condition) or the sum across blocks. Although ACoA patients and controls did not differ in their performance on the 0-s delay trial of the VCT task, they did differ significantly in their performance on the other delay trials (3 s, 9 s, and 18 s combined), t (42) = 2.09, p < .05…”

Comparison of figural intrusion errors in three amnesic subgroups

“…Thus, it must have been the two deficits combined that caused the increased number of intrusion errors. These findings are consistent with other reports indicating that in both Korsakoff patients (Freedman & Cermak, 1986) and ACoA patients (DeLuca, 1993), this same combination of deficits produced the greatest impairment.…”

Cognitive impairment following frontal lobe damage and its relevance to human amnesia.

“…with frontal lobe lesions tested in the present study did exhibit release from proactive interference. One possibility is that the impairment observed in KorsakofTs syndrome is the result of combined damage to frontal and diencephalic regions, as suggested by Freedman and Cermak (1986). Taken together, the findings from our study suggest that certain features of KorsakofTs syndrome may be attributed to frontal lobe dysfunction.…”

Neuropsychological correlates of short-term memory distractor tasks among patients with surgical repair of anterior communicating artery aneurysms.

“…ACoA patients did not differ significantly from controls on any of the dependent measures stemming from the release-from-PI task (see Table 3). Furthermore, when analyses identical to those performed by Freedman and Cermak (1986) were used, no differences in degree of release (Trial 5 − Trial 4) were found between ACoAs and controls on either the first block (first shift condition) or the sum across blocks. Although ACoA patients and controls did not differ in their performance on the 0-s delay trial of the VCT task, they did differ significantly in their performance on the other delay trials (3 s, 9 s, and 18 s combined), t (42) = 2.09, p < .05…”