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Cited by 29 publications
(26 citation statements)
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References 36 publications
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“…Summarizing several earlier studies, it would seem that it is difficult to definitively conclude that there is a correlation between the TNFa -308 polymorphism and heart failure (Ito et al, 2000;Kubota et al, 1998;Mishra et al, 2013;Vadlamani & Iyengar, 2004). Our data is consistent with these findings, although beside the lack of primary association, we showed a strong interaction between the TNFa -308 genotype and ApoAI level.…”
Section: Discussionsupporting
confidence: 91%
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“…Summarizing several earlier studies, it would seem that it is difficult to definitively conclude that there is a correlation between the TNFa -308 polymorphism and heart failure (Ito et al, 2000;Kubota et al, 1998;Mishra et al, 2013;Vadlamani & Iyengar, 2004). Our data is consistent with these findings, although beside the lack of primary association, we showed a strong interaction between the TNFa -308 genotype and ApoAI level.…”
Section: Discussionsupporting
confidence: 91%
“…The human TNFa gene is located on the chromosomal region 6p21.1-21.3, adjacent to the major histocompatibility complex, and in the neighborhood of genes playing role in the immune system like HLA, Hsp70 or the complement factor C4 (classical pathway) and factor B (alternative pathway). Moreover TNFa -308 is a part of the so-called 8.1 ancestral haplotype (8.1AH), the most frequent ancestral (Appoloni et al, 2004;Ito et al, 2000;Kubota et al, 1998;Mishra, Srivastava, Mittal, Garg, & Mittal, 2013), no primer correlation was found between the genotype and TNFa level in HF population, the above strong and conserved associations between the TNF -308 Moreover ApoAI is a negative acute phase protein, which level decreases in response to inflammation as a part of the acute phase reaction, and it can reflect the grade of inflammation (Khovidhunkit et al, 2004).…”
Section: Discussionmentioning
confidence: 99%
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“…We examined the effects of the NF-κB1 SNPs and predicted haplotypes of the LD block of the NF-κB1 gene on the genetic susceptibility to CAD in a Chinese Han population. During the past decade, much evidence has accumulated to support the role of NF-κB in CAD (Baker et al, 2009;Mishra et al, 2013). Our results provide genetic evidence that NF-κB1 is linked to CAD and extends to identify polymorphisms that may affect the development of CAD (Özbilüm et al, 2013;Yang et al, 2014;Arslan et al, 2015).…”
Section: Discussionmentioning
confidence: 52%
“…Polymorphisms of the NFKB1 gene have been implicated in susceptibility to left ventricular dysfunction and heart failure. 20,21 Other differentially regulated networks, centered around HLA-DR and ubiquitin C may suggest important differences in antigen recognition and intracellular parasitic protein amastigote assembly and degradation and may increase immune recognition of cardiac oxidized antigens. 22,23 However, differences in HLA-DR might be a reflection of its great genetic variability and therefore, difficult to interpret.…”
Section: Discussionmentioning
confidence: 99%