2019
DOI: 10.33263/briac92.874883
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Abstract: Cell membranes have unique features to store bio-energy in the physiology subjects. This work demonstrates a model of biological capacitors in the phospholipids bilayers membrane including DPPC, DOPG, DOPE, DOPS and DMPC structures. The electron densities profiles, electron localization function (ELF) and local information entropies have been used for studyng the interaction of G-proteins with phospholipid bilayers. The quantum and columbic blockade effects in different sizes and thicknesses of the membrane ha… Show more

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Cited by 1 publication
(1 citation statement)
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“…Among them, AD is the most common neurodegenerative disorder, affecting more than 45 million individuals worldwide and is expected to reach 60 million by 2030 due to the increase in the elderly population [17,18]. AD is characterized by the progressive death of cholinergic neurons within the hippocampal and cortical regions, the consequent atrophy, abnormal neurotransmission and loss of synapses, and neurodegeneration [4,[18][19][20][21]. At molecular levels, the underlying mechanisms of AD involve the extracellular deposition of amyloid-β (Aβ) peptides, known as amyloid plaques, and the intracellular formation of hyperphosphorylated tau (Tubulin Associated Unit) protein aggregates, known as neurofibrillary tangles (NFTs), which subsequently induce oxidative stress, chronic neuroinflammation, neuron dysfunction, and neurodegeneration [4,10,17,19,22,23].…”
Section: Introductionmentioning
confidence: 99%
“…Among them, AD is the most common neurodegenerative disorder, affecting more than 45 million individuals worldwide and is expected to reach 60 million by 2030 due to the increase in the elderly population [17,18]. AD is characterized by the progressive death of cholinergic neurons within the hippocampal and cortical regions, the consequent atrophy, abnormal neurotransmission and loss of synapses, and neurodegeneration [4,[18][19][20][21]. At molecular levels, the underlying mechanisms of AD involve the extracellular deposition of amyloid-β (Aβ) peptides, known as amyloid plaques, and the intracellular formation of hyperphosphorylated tau (Tubulin Associated Unit) protein aggregates, known as neurofibrillary tangles (NFTs), which subsequently induce oxidative stress, chronic neuroinflammation, neuron dysfunction, and neurodegeneration [4,10,17,19,22,23].…”
Section: Introductionmentioning
confidence: 99%