1988
DOI: 10.1001/archderm.124.9.1442b
|Get access via publisher |Summarize |Cite
|
Sign up to set email alerts

Purpura fulminans and transient protein C and S deficiency

Search citation statements

Order By: Relevance

Paper Sections

Select...
7
1
0
0

Citation Types

0
1
0
0

Year Published

1992
1992
2021
2021

Publication Types

Select...
7
1

Relationship

0
8

Authors

Journals

citations

Cited by 8 publications

(1 citation statement)
references

References 0 publications

0
1
0
0
Order By: Relevance
“…Interestingly, the most frequent viral infection associated with acquired purpura fulminans is varicella [42]. In two patients with this illness, reduced levels of protein C and protein S have been detected during the acute phase that became normal in the convalescence [45,46]. Recent reports have described a boy with varicella, purpura fulminans, a lupus anticoagulant and transitory severe protein S deficiency [48], and the occurrence, during recovery from varicella, of severe thromboembolic disease in a boy with a transient isolated deficiency of protein S due to the presence of a circulating autoantibody to protein S [41].…”
Section: Discussionmentioning
confidence: 99%
Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task.
“…Interestingly, the most frequent viral infection associated with acquired purpura fulminans is varicella [42]. In two patients with this illness, reduced levels of protein C and protein S have been detected during the acute phase that became normal in the convalescence [45,46]. Recent reports have described a boy with varicella, purpura fulminans, a lupus anticoagulant and transitory severe protein S deficiency [48], and the occurrence, during recovery from varicella, of severe thromboembolic disease in a boy with a transient isolated deficiency of protein S due to the presence of a circulating autoantibody to protein S [41].…”
Section: Discussionmentioning
confidence: 99%
Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task.
Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task.
Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task.