2005
DOI: 10.1289/ehp.7511
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Abstract: Most particulate matter (PM) health effects studies use outdoor (ambient) PM as a surrogate for personal exposure. However, people spend most of their time indoors exposed to a combination of indoor-generated particles and ambient particles that have infiltrated. Thus, it is important to investigate the differential health effects of indoor- and ambient-generated particles. We combined our recently adapted recursive model and a predictive model for estimating infiltration efficiency to separate personal exposu… Show more

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Cited by 184 publications
(155 citation statements)
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“…Season-specific models are also supported by the strong relationship between F inf and air exchange rates, which in turn are seasonal dependent (Janssen et al, 2002;Wallace et al, 2003;Koenig et al, 2005;Strand et al, 2006).…”
Section: Infiltration (F Inf ) Modeling Resultsmentioning
confidence: 86%
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“…Season-specific models are also supported by the strong relationship between F inf and air exchange rates, which in turn are seasonal dependent (Janssen et al, 2002;Wallace et al, 2003;Koenig et al, 2005;Strand et al, 2006).…”
Section: Infiltration (F Inf ) Modeling Resultsmentioning
confidence: 86%
“…However, although F inf has now been incorporated in a small number of health panel studies (Ebelt et al, 2005;Koenig et al, 2005;Strand et al, 2006;Allen et al, 2008), the effort and cost involved in collecting particle concentration data inside and outside homes have prevented large epidemiology studies from incorporating F inf in their exposure assessments.…”
Section: Introductionmentioning
confidence: 99%
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“…Among them, particulate air pollution, which has progressively increased in the last few decades, seems to be a quite plausible candidate as a major environmental determinant of asthma, as the increased incidence of asthma has been found to be strongly associated with exposure to particulate matter of the environment in many countries. [8][9][10][11] Moreover, air pollutants, such as particles, diesel exhaust, polycyclic aromatic hydrocarbons (PAHs), ozone, oxides of nitrogen, sulfur dioxide etc, have been shown to induce oxidative stress that was found to have an essential role in allergic inflammation, increasing airway responsiveness and inducing acute asthma exacerbations. 8,10,12 Several genetic studies have observed that individuals possessing loss-of-function variants of genes for glutathione S-transferases (antioxidant enzymes) are more susceptible to adjuvant effects of airborne pollutants increasing the risk of bronchial asthma.…”
Section: Introductionmentioning
confidence: 99%
“…[8][9][10][11] Moreover, air pollutants, such as particles, diesel exhaust, polycyclic aromatic hydrocarbons (PAHs), ozone, oxides of nitrogen, sulfur dioxide etc, have been shown to induce oxidative stress that was found to have an essential role in allergic inflammation, increasing airway responsiveness and inducing acute asthma exacerbations. 8,10,12 Several genetic studies have observed that individuals possessing loss-of-function variants of genes for glutathione S-transferases (antioxidant enzymes) are more susceptible to adjuvant effects of airborne pollutants increasing the risk of bronchial asthma. 13,14 We have recently confirmed that some other antioxidant genes are also proper candidates for asthma susceptibility genes.…”
Section: Introductionmentioning
confidence: 99%