Pruritus and Hyperesthesia Caused by Partial Sensory Denervation

Aubrun, Enrique A.

doi:10.1001/archderm.1936.01470160011002

Arch Derm Syphilol

1936

DOI: 10.1001/archderm.1936.01470160011002

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Pruritus and Hyperesthesia Caused by Partial Sensory Denervation

Enrique A. Aubrun¹

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Cited by 7 publications

(2 citation statements)

References 2 publications

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“…When he examined the skin's histology, he identified hair follicle atrophy, while the sweat and sebaceous glands were unaffected . These studies were not universally accepted however, and Aubrun concluded that the alopecia was merely a consequence of scratching . A more intriguing variation on the neuropathic theory was put forward by Jacquet.…”

Section: Alopecia Areata As An Atrophic Nerve Disordermentioning

confidence: 99%

“…[93,94] These studies were not universally accepted however, [95] and Aubrun concluded that the alopecia was merely a consequence of scratching. [96] A more intriguing variation on the neuropathic theory was put forward by Jacquet. He suggested AA was initiated by nerve irritation caused by defective and diseased teeth.…”

Section: Alopecia Are Ata a S An Atrophi C Nerve Disordermentioning

confidence: 99%

See 1 more Smart Citation

A “hair‐raising” history of alopecia areata

Broadley

¹

,

McElwee

²

2020

Experimental Dermatology

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Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task.

“…When he examined the skin's histology, he identified hair follicle atrophy, while the sweat and sebaceous glands were unaffected . These studies were not universally accepted however, and Aubrun concluded that the alopecia was merely a consequence of scratching . A more intriguing variation on the neuropathic theory was put forward by Jacquet.…”

Section: Alopecia Areata As An Atrophic Nerve Disordermentioning

confidence: 99%

“…[93,94] These studies were not universally accepted however, [95] and Aubrun concluded that the alopecia was merely a consequence of scratching. [96] A more intriguing variation on the neuropathic theory was put forward by Jacquet. He suggested AA was initiated by nerve irritation caused by defective and diseased teeth.…”

Section: Alopecia Are Ata a S An Atrophi C Nerve Disordermentioning

confidence: 99%

A “hair‐raising” history of alopecia areata

Broadley

¹

,

McElwee

²

2020

Experimental Dermatology

Get access via publisher Add to collection Cite

Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task.

Relationship Between Follicular Nerve Supply and Alopecia

Hordinsky

¹

,

Ericson

²

1996

Dermatologic Clinics

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Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task.

Die Haare

Richter¹

1962

Normale Und Pathologische Physiologie Der Haut I

3

0

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Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task.

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