Prostaglandin E2 accelerates sexual behavior in male rats

Cloprostenol enhances sexual behaviour and semen quality in growing lambs more effectively than Dinoprost

“…In rats, there are some pioneer studies using PGE2, but it should be considered that the mechanisms of action might differ from those of PGF2 and its analogues. Blumberg (1991) provoked increases in hypothalamic temperature associated with a reduction in the post‐ejaculatory refractoriness after administering PGE2 directly into the cerebral aqueduct. Moreover, castrated rats treated with testosterone increased more their number of copulas when PGE2 was intracranially administrated (Clemens & Glaude, 1977).…”

Cloprostenol enhances sexual behaviour and semen quality in growing lambs more effectively than Dinoprost

“…In rats, there are some pioneer studies using PGE2, but it should be considered that the mechanisms of action might differ from those of PGF2 and its analogues. Blumberg (1991) provoked increases in hypothalamic temperature associated with a reduction in the post‐ejaculatory refractoriness after administering PGE2 directly into the cerebral aqueduct. Moreover, castrated rats treated with testosterone increased more their number of copulas when PGE2 was intracranially administrated (Clemens & Glaude, 1977).…”

Cloprostenol enhances sexual behaviour and semen quality in growing lambs more effectively than Dinoprost

“…In rats, there are some pioneer studies using PGE2, but it should be considered that the mechanisms of action might differ from those of PGF2 and its analogues. Blumberg (1991) provoked increases in hypothalamic temperature associated with a reduction in the post‐ejaculatory refractoriness after administering PGE2 directly into the cerebral aqueduct. Moreover, castrated rats treated with testosterone increased more their number of copulas when PGE2 was intracranially administrated (Clemens & Glaude, 1977).…”

Cloprostenol enhances sexual behaviour and semen quality in growing lambs more effectively than Dinoprost

“…In rats, there are some pioneer studies using PGE2, but it should be considered that the mechanisms of action might differ from those of PGF2 and its analogues. Blumberg (1991) provoked increases in hypothalamic temperature associated with a reduction in the post‐ejaculatory refractoriness after administering PGE2 directly into the cerebral aqueduct. Moreover, castrated rats treated with testosterone increased more their number of copulas when PGE2 was intracranially administrated (Clemens & Glaude, 1977).…”