1996
DOI: 10.1080/01619569609595137
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Abstract: Since its inception, the John F. Kennedy Center has attempted to overcome developmental problems, which create restrictive barriers to the participation of individuals with specific disabilities in our broader society. Some of Nicholas Hobbs's earliest efforts involved developing strategies for preventing children's emotional and behavior problems, which interfered with their later full participation in society. Other investigators in the Kennedy Center explored ways of reducing dysfunctional repetitive moveme… Show more

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Cited by 15 publications
(14 citation statements)
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“…Prader±Willi syndrome is characterized by infantile hypotonia, hypogonadism, feeding difficulties, early childhood obesity, short stature, small hands and feet, intellectual disability and characteristic facial abnormalities (Cassidy 1984;Butler 1990; Thompson et al 1996;Cassidy et al 1997). Prader±Willi syndrome most often results from a paternal deletion of 15q11±q13 (in about 70% of cases), or sometimes, from maternal disomy of chromosome 15 (in about 30% of cases) (Ledbetter et al 1981;Butler et al 1986;Mascari et al 1992;Nicholls 1993;Cassidy et al 1997).…”
Section: Introductionmentioning
confidence: 99%
See 1 more Smart Citation
“…Prader±Willi syndrome is characterized by infantile hypotonia, hypogonadism, feeding difficulties, early childhood obesity, short stature, small hands and feet, intellectual disability and characteristic facial abnormalities (Cassidy 1984;Butler 1990; Thompson et al 1996;Cassidy et al 1997). Prader±Willi syndrome most often results from a paternal deletion of 15q11±q13 (in about 70% of cases), or sometimes, from maternal disomy of chromosome 15 (in about 30% of cases) (Ledbetter et al 1981;Butler et al 1986;Mascari et al 1992;Nicholls 1993;Cassidy et al 1997).…”
Section: Introductionmentioning
confidence: 99%
“…Intellectual disability and related adaptive behaviour deficits are common in PWS, although some relative strengths in self-help skills have been noted (Holm 1981;Thompson et al 1996). In addition, people with PWS show problem behaviours, including stubbornness, demanding attention and arguing (Taylor 1988;Dykens et al 1992).…”
Section: Introductionmentioning
confidence: 99%
“…Prader-Willi syndrome (PWS) is a genetic disorder characterized by infantile hypotonia and feeding difficulty, hypogonadism, early onset of obesity secondary to hyperphagia, mild intellectual disability, short stature, a particular facial appearance, and a paternal chromosome 15q11-q13 deletion in the majority of cases (Butler et al 1986;Butler 1990; Thompson et al 1996). The prevalence of obsessive-compulsive disorder (OCD) among people with mild to profound intellectual disability has been reported to be 3.5% (Vitiello et al 1989).…”
Section: Introductionmentioning
confidence: 99%
“…The clinical course of PWS includes two distinct, characteristic stages: the stage of neonatal and early infant development when the child is diagnosed with varying degrees of central hypotonia, hypogenitalism, sucking reflex disorders, and feeding difficulties resulting in poor weight gain (“failure to thrive”, FTT) (stage 1). The second stage entails the emergence of, among others, retardation of psychomotor development, a low level of physical activity, as well as behavioural feeding-related disorders with hyperphagic features, associated with the occurrence of early onset obesity (obesity is revealed on average at the age of 2) (stage 2) [ 11 , 13 , 15 ]. The transition from stage one to stage two, i.e.…”
Section: Introductionmentioning
confidence: 99%