1977
DOI: 10.1001/archotol.1977.00780290061007
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Posttraumatic Endolymphatic Hydrops

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Cited by 29 publications

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“…Secondary hydrops due to head trauma is thought to be one of the causes of sensorineural hearing loss (23,24). In our study, endolymphatic hydrops was localized to the apical turn.…”
Section: Discussionmentioning
confidence: 52%
Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task.
“…Secondary hydrops due to head trauma is thought to be one of the causes of sensorineural hearing loss (23,24). In our study, endolymphatic hydrops was localized to the apical turn.…”
Section: Discussionmentioning
confidence: 52%
Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task.
“…Although this is conjecture, it seems plausible since reduced physical energy or mental motivation can inhibit compensation (Bailey et al, 1993 ;Sullivan et al, 1993). Animal and human studies provide evidence that intense acoustic stimulation can result in damage to cells responsible for production or absorption of endolymph or disruption of the endolymphatic sac (Clark and Rees, 1977 ;Paparella and Mancini, 1983 ;Ylikoski, 1988 ;Kumagami, 1992). Of particular interest is that intense infrasound (<20 Hz), which is heavily weighted in a bomb explosion, produced hydrops in chinchilla (Lim et al, 1982) .…”
Section: Discussionmentioning
confidence: 99%
Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task.
“…As to post traumatic endolymphatic hydrops, there are several clinical reports. Pulec (1979), Clark and Thomas (1977), Ishikawa and Nakanishi (1983) reported cases of post-traumatic sensorineural hearing loss with dizziness. In particular Clark and Ishikawa reported that the glycerol test was positive and the deafness improved on treatment with diuretics.…”
Section: Discussionmentioning
confidence: 99%
Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task.