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Cited by 1,891 publications
(1,617 citation statements)
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References 32 publications
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“…These results show that, in contrast to the anti‐proliferative effects of FUT9 activity in the bulk of colon cancer cells (Fig 3A–D), FUT9 activity may actually be required for the efficient expansion of TIC populations. This was further confirmed by flow cytometry analysis, showing that FUT9 silencing decreases the expression of a prominent colorectal cancer TIC marker CD44 (Dalerba et al , 2007; Qureshi‐Baig et al , 2017) in HCT116 cells (Fig 4C).…”
Section: Resultssupporting
confidence: 56%
“…These results show that, in contrast to the anti‐proliferative effects of FUT9 activity in the bulk of colon cancer cells (Fig 3A–D), FUT9 activity may actually be required for the efficient expansion of TIC populations. This was further confirmed by flow cytometry analysis, showing that FUT9 silencing decreases the expression of a prominent colorectal cancer TIC marker CD44 (Dalerba et al , 2007; Qureshi‐Baig et al , 2017) in HCT116 cells (Fig 4C).…”
Section: Resultssupporting
confidence: 56%
“…Initially, it was believed that CSCs only comprise a small fraction in whole tumor cell populations, such as reported in solid and haematopoietic cancers (Al-Hajj et al, 2003;Jamieson et al, 2004;Singh et al, 2004;Dalerba et al, 2007;O/'Brien et al, 2007). Recent reports, based on transplantation of a broad spectrum of both primary and xenografted melanomas, contradict those conclusions (Quintana et al, 2008;Boiko et al, 2010).…”
Section: Discussionmentioning
confidence: 99%
“…Cancer cell heterogeneity and hierarchy may be resulted from both genetic and/or epigenetic causes. Increasing evidence from hematopoietic malignancies and solid tumors (including brain, breast, colorectal, head and neck cancers) has strongly supported the concept that a subpopulation of cancer cells in each tumor has greater potential of cancer initiation and repopulation (Lapidot et al, 1994;Bonnet and Dick, 1997;Al-Hajj et al, 2003;Hemmati et al, 2003;Singh et al, 2003;Galli et al, 2004;Singh et al, 2004;Ricci-Vitiani et al, 2007;O'Brien et al, 2007;Dalerba et al, 2007;Prince et al, 2007;Schatton et al, 2008). These cells were called cancer stem cells (CSCs) or tumor-initiating or propagating cells because they share some critical characteristics with normal stem cells, including the capacities for self-renewal, multi-lineage differentiation, and maintained proliferation (Reya et al, 2001;Vescovi et al, 2006;Bao et al, 2006a;Rosen and Jordan, 2009;Park and rich, 2009;Heddleston et al, 2010;Frank et al, 2010).…”
Section: Introductionmentioning
confidence: 99%