1979
DOI: 10.1001/archderm.115.4.446
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Pemphigoid vegetans

Abstract: A 24-year-old woman had massive purulent and verrucous vegetations with fissuring in the groin and axillae. She also had scaling, crusted, bullous, and purulent lesions on the scalp, face, axillae, umbilicus, groin, and hands. Biopsy specimens of skin for histopathologic examinations and direct immunofluorescent study showed typical bullous pemphigoid, and serial serum samples tested by indirect immunofluorescent study confirmed the diagnosis. A basement membrane antibody was presented on four occasions at tit… Show more

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Cited by 20 publications
(17 citation statements)
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“…Circulating BMZ antibodies are usually demonstrated in 70-80% of patients with bullous pemphigoid (Hodge et al, 1978b); these antibodies are specific for pemphigoid, and react with all stratified epithelia. [Recently it has been suggested that these pemphigoid antibodies can be divided into two classes on the basis of the demonstration of antibody binding to the cell membrane and cytoplasm of normal human basal cells, in addition to tbe BMZ binding antibody (Bean et al, 1976) Desquamative gingivitis (Rogers, Sheridan & Jordan, 1976) Localized pemphigoid (Brunsting & Perry, 1957;Sparrow & Moynahan, 1976) Pemphigoid vegetans (Winkelmann & Su, 1979) Pemphigoid nodularis (Yung, Soltani & Lorincz, 1981) 'Drug'-induced pemphigoid: e.g. UVL-induced pemphigoid (Person & Rogers, 1976) PUVA-induced pemphigoid (Thompson & Schmidt, 1976;Abel & Bennett, 1979) Frusemide-induced pemphigoid (Ebringer, Adam & Parkin 1969;Fellner & Katz, 1976) Figure 5.…”
Section: Imf Findings In the Bullous Disordersmentioning
confidence: 99%
“…Circulating BMZ antibodies are usually demonstrated in 70-80% of patients with bullous pemphigoid (Hodge et al, 1978b); these antibodies are specific for pemphigoid, and react with all stratified epithelia. [Recently it has been suggested that these pemphigoid antibodies can be divided into two classes on the basis of the demonstration of antibody binding to the cell membrane and cytoplasm of normal human basal cells, in addition to tbe BMZ binding antibody (Bean et al, 1976) Desquamative gingivitis (Rogers, Sheridan & Jordan, 1976) Localized pemphigoid (Brunsting & Perry, 1957;Sparrow & Moynahan, 1976) Pemphigoid vegetans (Winkelmann & Su, 1979) Pemphigoid nodularis (Yung, Soltani & Lorincz, 1981) 'Drug'-induced pemphigoid: e.g. UVL-induced pemphigoid (Person & Rogers, 1976) PUVA-induced pemphigoid (Thompson & Schmidt, 1976;Abel & Bennett, 1979) Frusemide-induced pemphigoid (Ebringer, Adam & Parkin 1969;Fellner & Katz, 1976) Figure 5.…”
Section: Imf Findings In the Bullous Disordersmentioning
confidence: 99%
“…In the six previously reported cases of pemphigoid vegetans, the age of onset ranged from 23 to 82 years (Table 1). Pemphigoid vegetans has been associated with inflammatory bowel disease in two cases 1,2 . Mucosal involvement was identified in five of six cases, and serum eosinophilia was a documented feature in two cases (Table 1).…”
Section: Discussionmentioning
confidence: 97%
“…Pemphigoid vegetans is a rare intertriginous variant of bullous pemphigoid characterized by vegetative and purulent lesions present in the groin, axillae, thighs, hands, eyelids and perioral regions. Originally described in 1979 by Winkelmann and Su, 1 pemphigoid vegetans is exceptionally rare, with only six cases published in the English literature 1–6 . Pemphigoid vegetans has been described as an autoimmune disorder with autoantibodies directed against basal cell hemidesmosomes.…”
mentioning
confidence: 99%
“…There are several distinct clinical presentations of BP, which have so far been described: generalized bullous form, vegetative form [19,20], vesicular form [21], generalized erythrodermic form [22,23], urticarial form, localized (usually pretibial) form [2,24] and nodular form [25,26,27,28]. …”
Section: Introductionmentioning
confidence: 99%