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Cited by 8 publications
(5 citation statements)
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References 7 publications
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“…In addition, we found that the expression of CIT-K gene was positively correlated with TP63 gene in normal breast tissue, but negatively correlated with TP63 gene in tumor tissue. P63 is mainly expressed in the nuclei of myoepithelial cells of normal breast ducts and lobules and some special pathological type of breast cancer, such as metaplastic breast carcinomas, breast sarcomas and medullary breast cancer, while the positive rate of P63 in invasive breast cancer is extremely low [30]. Therefore, we speculate that the CIT-K is also higher in cancer cells with higher expression of Ki67, but the relationship between the expression of P63 and CIT-K needs to be further studied.…”
Section: Discussionmentioning
confidence: 99%
“…In addition, we found that the expression of CIT-K gene was positively correlated with TP63 gene in normal breast tissue, but negatively correlated with TP63 gene in tumor tissue. P63 is mainly expressed in the nuclei of myoepithelial cells of normal breast ducts and lobules and some special pathological type of breast cancer, such as metaplastic breast carcinomas, breast sarcomas and medullary breast cancer, while the positive rate of P63 in invasive breast cancer is extremely low [30]. Therefore, we speculate that the CIT-K is also higher in cancer cells with higher expression of Ki67, but the relationship between the expression of P63 and CIT-K needs to be further studied.…”
Section: Discussionmentioning
confidence: 99%
“…P63 expression has been recommended as a reliable indicator of histologic grade and prognosis in breast cancer, meningioma and OSCC. It may be due to oncogenic role of one p63 isotype, ΔNP63, in proliferation and differentiation [19][20][21]. Additionally, p63 has been demonstrated as a helpful marker in distinguishing low-grade MEC from papillary cystadenoma of salivary glands [1].…”
Section: Discussionmentioning
confidence: 99%
“…Все большую актуальность приобретает изучение биологических маркеров РМЖ, альтернативных стандартным маркерам, широко вошедшим в рутинную клиническую практику. Целый ряд показателей, таких как р53, цитокератины (cytokeratin, CK) 5 и 6 (CK5/6), гладкомышечный актин (SMA), p63, PHH3, E-кадгерин, EGFR, FOXA1, рецепторы андрогенов (РА), TILs и др., в многочисленных исследованиях демонстрируют свою предиктивную и / или прогностическую значимость [12][13][14][15][16][17][18][19][20][21]. Результаты данных исследований свидетельствуют о том, что изучение новых биологических маркеров при РМЖ требует дальнейшего подробного анализа.…”
Section: обзорные статьиunclassified
“…Актуальные исследования свидетельствуют, что ключевую роль в развитии резистентности играет мутация гена рецептора эстрогенов ESR1. Большинство мутаций М а м м о л о г и я / Том 15 / Vol 15.…”
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