2016
DOI: 10.1097/wnr.0000000000000588 View full text |Buy / Rent full text
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Abstract: Neuronal death is a pathophysiological process that is often caused by hypoxia/ischemia. However, the causes of hypoxia/ischemia-induced neuronal death are debated, and additional experimental data are needed to resolve this debate. In the present study, we applied oxygen-glucose deprivation (OGD) to PC12 cells and hippocampal neurons to establish a hypoxia/ischemia model. We evaluated the effects of OGD on cell death/apoptosis and on the levels of two excitatory amino acid neurotransmitters, aspartic acid and… Show more

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“…Although the metabolic pathway is not entirely clear, it can inhibit the central nervous system (24). Aspartic acid (Asp) is a neurotransmitter in the central nervous system; it is unevenly distributed and has a strong excitatory effect on cerebral cortex cells (28). Studies on Asp and Gly during anaesthesia are limited, therefore the present study focused on xylazineinduced changes in Asp and Gly release during anaesthesia.…”
Section: Introductionmentioning
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“…Although the metabolic pathway is not entirely clear, it can inhibit the central nervous system (24). Aspartic acid (Asp) is a neurotransmitter in the central nervous system; it is unevenly distributed and has a strong excitatory effect on cerebral cortex cells (28). Studies on Asp and Gly during anaesthesia are limited, therefore the present study focused on xylazineinduced changes in Asp and Gly release during anaesthesia.…”
Section: Introductionmentioning