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Cited by 49 publications
(18 citation statements)
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“…Progestagen treatment resulted in inhibition of ovarian compensatory hyper¬ trophy in all experiments (Table 3), and prevented the compensatory increase in the number of corpora lutea usually seen in the unilaterally spayed rats (Table 3) (Table 4), confirming the previous reports (Edgren, Parlow, Peterson & Jones, 1965;Labhsetwar, 1967c). In contrast, the removal of both ovaries resulted in a several-fold increase in the pituitary stores of both gonadotrophins (Table 4).…”
Section: Ovarian Compensatory Hypertrophysupporting
confidence: 85%
“…Progestagen treatment resulted in inhibition of ovarian compensatory hyper¬ trophy in all experiments (Table 3), and prevented the compensatory increase in the number of corpora lutea usually seen in the unilaterally spayed rats (Table 3) (Table 4), confirming the previous reports (Edgren, Parlow, Peterson & Jones, 1965;Labhsetwar, 1967c). In contrast, the removal of both ovaries resulted in a several-fold increase in the pituitary stores of both gonadotrophins (Table 4).…”
Section: Ovarian Compensatory Hypertrophysupporting
confidence: 85%
“…No significant differences in FSH and LH of the pituitary gland were observed between intact and semispayed rats (2). Using an assay for total gonadotropins, no differences were detected between intact and unilaterally ovariectomized mice (1).…”
mentioning
confidence: 99%
“…It is alluded to relative increase-in the. availability of serum gonadotrophins to the remaining single ovary after hemispaying, since no increase in the pituitary gonadotrophins is observable after hemispaying (McLaren 1963(McLaren , 1966Edgren et al 1965). However, this contention is questioned as there is an increase in the gonadotrophin output, due to decrease in the circulating estrogen after hemispaying which is responsible for the ovarian compensatory hypertrophy (Grady and Greenwald 1968 ;Benson et al 1969;Walshen 1970Walshen , 1972Howland and Skinner 1973).…”
Section: 4 Estrous Cyclementioning
confidence: 99%
“…ovulation and hypertrophy in rats, mice and hamsters (Arai 1920 ;Mandl and Zuckerman 1951;Greenwald 1961 ;Pepler 1975). This may be due to the unchanged availability of pituitary gonadotrophins (FSH and LH) to the remaining single ovary and/or increase in the pituitary release of gonadotrophins due to decrease in the circulating estrogen after semiovariectomy (Edgren et at 1965 ;Welchen 1970Welchen , 1972Howland et al 1974). The compensatory response of the ovary may continue till the ovary gets the increased amount of pituitary gonadotrophins and once the gonadotrophins level falls down to normal, due to increase in the steroid output by the hypertrophied ovary, this compensatory hypertrophy may also decrease.…”
Section: Introductionmentioning
confidence: 99%