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citations
Cited by 340 publications
(304 citation statements)
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References 42 publications
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“…>10 g/l), long-term prophylactic administration of antibiotics is not necessary since the risk of SBP in these patients is negligible, provided adequate prophylaxis is administered if and when gastrointestinal haemorrhage develops in the course of the disease. Nonselective beta blockers increase the risk for hepatorenal syndrome and death in patients with cirrhosis and SBP [19] and should be carefully used.…”
Section: Resultsmentioning
confidence: 99%
“…>10 g/l), long-term prophylactic administration of antibiotics is not necessary since the risk of SBP in these patients is negligible, provided adequate prophylaxis is administered if and when gastrointestinal haemorrhage develops in the course of the disease. Nonselective beta blockers increase the risk for hepatorenal syndrome and death in patients with cirrhosis and SBP [19] and should be carefully used.…”
Section: Resultsmentioning
confidence: 99%
“…The choice of treatment should be based on local resources, expertise and contraindications [25,26]. However, regarding the use of beta blockers, a recent retrospective study by Mandorfer et al suggests that they are responsible for an increased hospitalization of hemodynamically compromised patients [27]. Acute hemorrhage from variceal rupture is well known to be an emergency in clinical practice.…”
Section: Varices and Bleedingmentioning
confidence: 99%
“…Ascites is the most common complication of cirrhosis, and about 60% of patients with compensated liver disease develop ascites during the clinical course of the disease [26]. Mortality is approximately 40% at 1 year, and 50% at 2 years [26][27][28][29][30][31]. The most reliable predictors of poor prognosis include hyponatremia, low arterial pressure, increased serum creatinine, and low urine sodium [31].…”
Section: Ascites and Spontaneous Bacterial Peritonitismentioning
confidence: 99%
“…It is in this pathophysiological context that beta-blockers have theoretical benefits, first by reducing cardiac output via beta-1 adrenergic blockade, and second, by reducing portal blood flow via beta-2 adrenergic blockade and splanchnic vasoconstriction [17] . Moreover, independently of their hemodynamic effects, treatment with betablockers decreased intestinal permeability and significantly reduced the risk of spontaneous bacterial peritonitis in both hemodynamic responders and non-responders [18] . The benefits of beta-blockade therapy were demonstrated in several solid studies as shown in table 1 below.…”
Section: Beta-blockers In Cirrhosismentioning
confidence: 98%
“…This condition was associated with shortened survival even though it may be clinically silent [49] . In addition, Mandorfer et al noticed an increased risk for hepatorenal syndrome and death in patients with cirrhosis and spontaneous bacterial peritonitis [18] . These findings led to a reconsideration of beta-blocking therapy in cirrhotic patients especially those with advanced disease and refractory ascites.…”
mentioning
confidence: 99%