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Cited by 275 publications
(234 citation statements)
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References 39 publications
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“…Nonselective β-blockers are primarily known to reduce portal pressure by reducing portal inflow. Otherwise, they have also been investigated in more detail under the rationale of reducing bacterial translocation [43]. The permeability index correlated with the hepatic venous pressure gradient and, under β-blocker therapy, not only was the hepatic venous pressure gradient decreased, but so too was intestinal permeability.…”
Section: Clinical Studiesmentioning
confidence: 99%
See 1 more Smart Citation
“…Nonselective β-blockers are primarily known to reduce portal pressure by reducing portal inflow. Otherwise, they have also been investigated in more detail under the rationale of reducing bacterial translocation [43]. The permeability index correlated with the hepatic venous pressure gradient and, under β-blocker therapy, not only was the hepatic venous pressure gradient decreased, but so too was intestinal permeability.…”
Section: Clinical Studiesmentioning
confidence: 99%
“…The permeability index correlated with the hepatic venous pressure gradient and, under β-blocker therapy, not only was the hepatic venous pressure gradient decreased, but so too was intestinal permeability. Abnormal gastroduodenal permeability and intestinal permeability was associated with a reduced risk for variceal bleeding, but no effect on mortality has been observed [43]. …”
Section: Clinical Studiesmentioning
confidence: 99%
“…The overgrowth of intestinal bacteria and an abnormal intestinal motility associated with portal hypertension are two factors favoring translocation and predisposing to SBP [9]. The other factor predisposing to SBP development is the immune deficiency typical of patients with advanced cirrhosis [10].…”
Section: Translocation and Sbp Pathogenesismentioning
confidence: 99%
“…22 Moreover, patients with cirrhosis and bacterial translocation have a higher degree of liver dysfunction (estimated by Child-Pugh score) in comparison with patients without bacterial translocation. 23 Reiberger et al 24 have reported a significant correlation between portal pressure and gastroduodenal and intestinal permeability, plasma levels of LPS-binding protein (LBP) and IL-6 in patients with cirrhosis and PHT. Patients with severe PHT (hepatic venous pressure gradient [HVPG] >20 mmHg had significantly increased markers of gastroduodenal/intestinal permeability (urine sucrose levels p=0.049; sucrose/mannitol ratios p=0.007; intestinal permeability indices p=0.002), and bacterial translocation than those having HVPG <20 mmHg.…”
Section: Tropical Gastroenterology 2013;34(3):119-122mentioning
confidence: 99%