Multiple intrahepatic pseudocysts in acute pancreatitis

Abstract

Liver pseudocysts are a very rare complication in acute pancreatitis with only a few cases previously described. The lack of experience and literature on this condition leads to difficulties in the differential diagnosis and management. We report herein a case of acute pancreatitis who developed multiple intrahepatic pseudocysts. After complete imaging evaluation, the diagnosis was still unclear and the patient was operated on. The presence of liver lesions in patients with acute pancreatitis should raise the possibility of intrahepatic pseudocysts.

Intrahepatic localization of pancreatic pseudocyst: A case report

“…Besides, percutaneous drainage, surgery, or ES can be performed (3,(9)(10)(11)(12)13). In the patient we reported here, percutaneous drainage was performed because the patient had abdominal pain and the size of the pseudocyts was 130 mm in length.…”

“…The second mechanism consists of spreading of pancreatic fluid from the head of the pancreas into the hepatoduodenal ligament and porta hepatis along the portal vein and its branches. This results in formation of intraparenchymal collections (6,8,9). Subcapsular pseudocysts are located just beneath the liver capsule and are biconvex in shape, while intraparenchymal pseudocysts are located away from the liver capsule and near the porta hepatis branches (5).…”

Intrahepatic pseudocyst complicating acute pancreatitis: A rare case report

“…In the usual temporal evolution a pancreatic pseudocyst requires a minimum of time, between 4 and 6 wk, to form the welldefined wall, a main characteristic of it. [9] However, the time span in our case extremely short. A possible explanation for this can be hat the patient may have suffered from a previous episode of pancreatitis and that the present episode might in fact be a relapsing pancreatitis.…”

Intrahepatic Pancreatic Pseudocyst Complicated by Pancreatitis: A Case Report

“…4,[10][11][12][13] The first mechanism is consisted of pancreatic enzyme released following acute pancreatitis into the lesser sac spreading along the lesser omentum and gastrohepatic ligaments. This mechanism explains why IHPPs are frequently observed in the left lobe.…”