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Cited by 84 publications
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“…12 AP-1 was shown to mediate shear-induced monocyte chemotactic protein-1 expression. 13 Further characterization of the role of these sites in the shear-induced eNOS expression is underway in our laboratory.…”
Section: Discussionmentioning
confidence: 99%
“…12 AP-1 was shown to mediate shear-induced monocyte chemotactic protein-1 expression. 13 Further characterization of the role of these sites in the shear-induced eNOS expression is underway in our laboratory.…”
Section: Discussionmentioning
confidence: 99%
“…26,58 Furthermore, multiple cis elements that mediate transcriptional regulation by shear stress have now been identified. 61,62 The robust nature of the Cx43 response to both stretch and shear implies that this gap junction protein is important in communicating alterations in mechanical environment in both endothelial cells and smooth muscle cells, and it may impart functional changes to these cells. Several investigators have proposed that changes in gap junctional activity contribute to the increased vascular reactivity that is observed in hypertension.…”
Section: Discussionmentioning
confidence: 99%
“…These include several proadhesive, proinflammatory, or prothrombotic genes such as intercellular adhesion molecule-1 (14), monocyte chemoattractant protein-1 (15), and tissue factor (TF) (16). Transcriptional activation of these genes has been shown to proceed via cis -acting sequences that bind transcription factors Sp1 (16), NF-B (17), AP1 (18,15), and early growth response-1 (Egr-1) (12,13). Transcription factors AP1 and NF-B are themselves upregulated by shear stress (19).…”
Section: Introductionmentioning
confidence: 99%