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citations
Cited by 117 publications
(103 citation statements)
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References 141 publications
(103 reference statements)
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“…In addition to the increased renal Ca 2+ excretion, polyuria and an acidic urine were consistently observed in TRPV5 −/− mice. Increasing the amount of urine and acidification of the urine reduce the potential risk of renal stone formation (Baumann 1998;Frick and Bushinsky 2003;Miller and Stapleton 1989). Micropunc-ture studies indicated that the renal Ca 2+ transport defect was localized primarily to the DCT/CNT.…”
Section: Trpv5 Knockout Micementioning
confidence: 99%
“…In addition to the increased renal Ca 2+ excretion, polyuria and an acidic urine were consistently observed in TRPV5 −/− mice. Increasing the amount of urine and acidification of the urine reduce the potential risk of renal stone formation (Baumann 1998;Frick and Bushinsky 2003;Miller and Stapleton 1989). Micropunc-ture studies indicated that the renal Ca 2+ transport defect was localized primarily to the DCT/CNT.…”
Section: Trpv5 Knockout Micementioning
confidence: 99%
“…The most common risk factor for nephrolithiasis is hypercalciuria, the excess excretion of urinary calcium (Coe et al 2005). We have used the genetic hypercalciuric stone-forming rat (GHS), a model of this phenotype developed from Sprague-Dawley (SD) rats selected for urinary calcium excretion (Li et al 1993;Bushinsky et al 1995Bushinsky et al , 2002Bushinsky et al , 2006Levy et al 1995;Krieger et al 1996;Tsuruoka et al 1997;Yao et al 1998;Frick and Bushinsky 2003), to map two quantitative trait loci (QTL) for urinary calcium excretion using F 2 intercrosses of GHS rats and normocalciuric Wistar-Kyoto (WKY) rats (Hoopes et al 2008): HC1 (on rat chromosome (RNO) 1; (Hoopes et al 2003), and HC2 (RNO4) (Scheinman et al 2008), both of which were detected only in females. These genes are underlain by an array of genes associated with calcium physiology (Hoopes et al 2006).…”
mentioning
confidence: 99%
“…In this respect, absorptive, renal, and resorptive hypercalciuria are distinguished: Disturbed intestinal Ca 2ϩ hyperabsorption, impaired renal Ca 2ϩ reabsorption, and increased bone resorption, respectively, are primarily involved. [3][4][5][6] Current treatment strategies of renal stones consist of (pain) medication, dietary adjustments, hydration, extracorporeal shock wave treatment, or surgery.…”
mentioning
confidence: 99%