Lung injury following exposure of rats to relatively high mass concentrations of nitrogen dioxide

Nitric Oxide As a Bactericidal Agent: Is the Cure Worse Than the Disease?

“…6 High concentrations of NO, on the other hand, may have deleterious effects, including acute pulmonary injury, methemoglobinemia, asphyxia, and death. [7][8][9] High levels of intracellular NO have been reported to cause DNA damage and mutations in human cell preparations. 10 The United States Occupational Safety and Health Administration has set a time-weighted average exposure limit of 25 ppm for NO when breathed for 8 h/d in the workplace.…”

Nitric Oxide As a Bactericidal Agent: Is the Cure Worse Than the Disease?

“…6 High concentrations of NO, on the other hand, may have deleterious effects, including acute pulmonary injury, methemoglobinemia, asphyxia, and death. [7][8][9] High levels of intracellular NO have been reported to cause DNA damage and mutations in human cell preparations. 10 The United States Occupational Safety and Health Administration has set a time-weighted average exposure limit of 25 ppm for NO when breathed for 8 h/d in the workplace.…”

Severe lung injury following inhalation of nitric acid fumes

“…[2] Nitric acid fumes and its decomposition gases such as oxides of nitrogen can cause devastating illness. [3][4][5] We are presenting this rare case of survival following de layed onset of severe pulmonary edema, resulting from accidental inhalation of nitric acid fumes.…”

Evaluation of NOX-Induced Toxicity

“…The RCL were oven-dried to a constant weight and reweighed (right cranial lobe dry weight, RCLDW). Inasmuch as the post-exposure changes in the LWW, RCLWW, and RCLDW values after N02 exposure usually paralleled one another, only the LWW data will be reported herein as an endpoint for characterizing the severity of N02-induced pulmonary edema Lehnert et al, 1993).…”

Nitric Oxide As a Bactericidal Agent: Is the Cure Worse Than the Disease?

“…6 High concentrations of NO, on the other hand, may have deleterious effects, including acute pulmonary injury, methemoglobinemia, asphyxia, and death. [7][8][9] High levels of intracellular NO have been reported to cause DNA damage and mutations in human cell preparations. 10 The United States Occupational Safety and Health Administration has set a time-weighted average exposure limit of 25 ppm for NO when breathed for 8 h/d in the workplace.…”

Severe lung injury following inhalation of nitric acid fumes

“…[2] Nitric acid fumes and its decomposition gases such as oxides of nitrogen can cause devastating illness. [3][4][5] We are presenting this rare case of survival following de layed onset of severe pulmonary edema, resulting from accidental inhalation of nitric acid fumes.…”

Evaluation of NOX-Induced Toxicity

“…The RCL were oven-dried to a constant weight and reweighed (right cranial lobe dry weight, RCLDW). Inasmuch as the post-exposure changes in the LWW, RCLWW, and RCLDW values after N02 exposure usually paralleled one another, only the LWW data will be reported herein as an endpoint for characterizing the severity of N02-induced pulmonary edema Lehnert et al, 1993).…”

Nitric Oxide As a Bactericidal Agent: Is the Cure Worse Than the Disease?

“…6 High concentrations of NO, on the other hand, may have deleterious effects, including acute pulmonary injury, methemoglobinemia, asphyxia, and death. [7][8][9] High levels of intracellular NO have been reported to cause DNA damage and mutations in human cell preparations. 10 The United States Occupational Safety and Health Administration has set a time-weighted average exposure limit of 25 ppm for NO when breathed for 8 h/d in the workplace.…”

Severe lung injury following inhalation of nitric acid fumes

“…[2] Nitric acid fumes and its decomposition gases such as oxides of nitrogen can cause devastating illness. [3][4][5] We are presenting this rare case of survival following de layed onset of severe pulmonary edema, resulting from accidental inhalation of nitric acid fumes.…”

Evaluation of NOX-Induced Toxicity

“…The RCL were oven-dried to a constant weight and reweighed (right cranial lobe dry weight, RCLDW). Inasmuch as the post-exposure changes in the LWW, RCLWW, and RCLDW values after N02 exposure usually paralleled one another, only the LWW data will be reported herein as an endpoint for characterizing the severity of N02-induced pulmonary edema Lehnert et al, 1993).…”