Investigation of Hyperparathyroidism in the Absence of Bone Disease

Hyde, R. D.; Jones, Ralph; McSwiney, R. R.; Prunty, F. T. G.

doi:10.1016/s0140-6736(60)90171-9

Cited by 51 publications

(16 citation statements)

References 16 publications

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“…In ours, the variability of [Ca2+] is greater than that of total calcium; in theirs, variability of the control series is low. Apart from these differences in variability, the two studies, as well as three other recent reports (34)(35)(36), are substantially in agreement in showing little or no difference in percentage ionization of plasma calcium in primary hyperparathyroidism as compared with normal.…”

Section: Discussionsupporting

confidence: 81%

The Separate Effects of Hyperparathyroidism, Hypercalcemia of Malignancy, Renal Failure, and Acidosis on the State of Calcium, Phosphate, and Other Ions in Plasma*

Walser¹

1962

J. Clin. Invest.

117

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Section: Discussionsupporting

confidence: 81%

The Separate Effects of Hyperparathyroidism, Hypercalcemia of Malignancy, Renal Failure, and Acidosis on the State of Calcium, Phosphate, and Other Ions in Plasma*

Walser¹

1962

J. Clin. Invest.

117

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“…12/60 autoanalyser. The renal phosphate threshold was assessed by a phosphate infusion technique (Hyde et al, 1960) using a continuously accelerating infusion pump. Alkaline …”

Section: Methodsmentioning

confidence: 99%

Osteomalacia after Prolonged Glutethimide Administration

Greenwood¹,

Prunty²,

Silver³

1973

BMJ

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SummaryVitamin D deficiency is a previously unreported complication of therapy with hypnotics, and we here report a case of osteomalacia associated with long-term glutethimide administration. There was evidence of pronounced hepatic enzyme induction, and the plasma half-time of 3H vitamin D, was decreased by this drug. In addition, raised levels of serum gamma-glutamyl transpeptidase, 5-nucleotidase, and leucine aminopeptidase were observed and the patient excreted large amounts ofxylulose. These changes were reversed by stopping the glutethimide.

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“…Les cas d'hyperparathyroïdie sans modification de la calcémie sont connus; il existe peut-être cependant, dans ces cas, une élévation du calcium ionisé du sang, bien que depuis lors plusieurs cas aient été décrits d'hvperparathyroïdie où le calcium ionisé du sang était également normal (Hyde [24]; McStviney [27]). …”

Section: Discussionunclassified