Search citation statements

Order By: Relevance

Paper Sections

Select...
3
1
1

Citation Types

5
64
0

Year Published

2019
2019
2023
2023

Publication Types

Select...
8

Relationship

1
7

Authors

Journals

citations
Cited by 80 publications
(69 citation statements)
references
References 171 publications
5
64
0
Order By: Relevance
“…MicroRNAs (miRNAs) are short RNA molecules that are unable to encode proteins, but which can still exert biological effects, suppressing or promoting the progression of cancers in a tumor-and miRNAspecific manner via mediating the posttranscriptional suppression and/or degradation of target mRNAs [4]. A wide number of miRNAs have recently been shown to serve vital roles in many cancer types including CRC [5], serving as potential diagnostic, prognostic, or therapeutic biomarkers in CRC. Multiple past studies have demonstrated the ability of miR-365 to suppress the progression of cutaneous squamous cell carcinoma [6], lung [7], breast [8], and hepatic carcinoma [9].…”
Section: Ivyspringmentioning
confidence: 99%
“…MicroRNAs (miRNAs) are short RNA molecules that are unable to encode proteins, but which can still exert biological effects, suppressing or promoting the progression of cancers in a tumor-and miRNAspecific manner via mediating the posttranscriptional suppression and/or degradation of target mRNAs [4]. A wide number of miRNAs have recently been shown to serve vital roles in many cancer types including CRC [5], serving as potential diagnostic, prognostic, or therapeutic biomarkers in CRC. Multiple past studies have demonstrated the ability of miR-365 to suppress the progression of cutaneous squamous cell carcinoma [6], lung [7], breast [8], and hepatic carcinoma [9].…”
Section: Ivyspringmentioning
confidence: 99%
“…Increasing numbers of studies have identified that lncRNA, microRNA (miRNA), and protein-coding message RNA (mRNA) compose complex RNA networks by competitive association. Abnormal regulation or aberrant expression of various lncRNA plays a role in the initiation, progression, and relapse of colorectal cancer (2,3).…”
Section: Introductionmentioning
confidence: 99%
“…52 The main characteristics of this transformation are decreased expression of cell adhesion Hepatocellular carcinoma TNM stage, metastasis [29] Tumor size, TNM stage, ALT (U/L) [14] Shorter DFS and OS, multiple tumors, macro vascular invasion [26] Shorter DFS and OS [28] Lymph node involvement, TNM stage [27] Tumor size, TNM stage, vascular invasion [25] Tumor size, TNM stage, metastasis, portal vein tumor thrombus (PVTT) [30] Cervical DovePress molecules (such as E-cadherin), vimentin, rather than cytokeratin, as the main cytoskeletal protein, and the cells taking on the morphological characteristics of mesenchymal cells. 53 This phenotypic plasticity, named epithelial-to-mesenchymal transition (EMT), causes epithelial cells to lose cell polarity, lose the connection with basement membrane and other epithelial phenotypes, and gain higher migration and invasion ability. 52 Several studies have shown that the overexpression of SNHG16 can promote EMT and migration, thus promoting the invasion of cancer cells.…”
Section: Activating Migration and Invasionmentioning
confidence: 99%