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Cited by 69 publications
(41 citation statements)
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References 55 publications
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“…Administration of 50 and 100 ng CART (54-102) to normal rats by the icv route produced significant anxiety-like behavior in the social interaction test. The peptide was also given at lower doses (10 and 20 ng/rat) directly into the amygdala because this is believed to be the locus for generation of anxiety-like behavior (Adolphs et al, 1994;Kokare et al, 2005), and produced a similar response. The data suggest that CeA might be the site of anxiogenic-like action of CART.…”
Section: Discussionmentioning
confidence: 99%
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“…Administration of 50 and 100 ng CART (54-102) to normal rats by the icv route produced significant anxiety-like behavior in the social interaction test. The peptide was also given at lower doses (10 and 20 ng/rat) directly into the amygdala because this is believed to be the locus for generation of anxiety-like behavior (Adolphs et al, 1994;Kokare et al, 2005), and produced a similar response. The data suggest that CeA might be the site of anxiogenic-like action of CART.…”
Section: Discussionmentioning
confidence: 99%
“…Both peptides are anorectic (Tian et al, 2004) and implicated in the regulation of anxiety-like behavior, depression, and stress like conditions (Kuhar et al, 2002;Chaki et al, 2003;Kokare et al, 2005;Goyal et al, 2006;Stanek, 2006). In addition, a-MSH has anxiogenic properties (Kokare et al, 2005) and increases in association with ethanol withdrawal-induced anxiety-like behavior . Furthermore, administration of antibodies against a-MSH enhances the anxiolytic effect of ethanol .…”
Section: Discussionmentioning
confidence: 99%
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“…Recently it was discovered that optogenetic activation of ARC AgRP-neurons leads to aversion, and that these neurons directly respond to cues associated with food by decreasing their activity (Betley et al, 2015). Other studies have implied a role for POMC-derived melanocortins in signaling negative affect, such as anxiety and depression (Lim et al, 2012;Liu et al, 2013;Goyal et al, 2006;Kokare et al, 2005). Intracerebroventricular (i.c.v) or intra-amygdala injections of melanocortins in the CNS triggers stress signaling, including HPA-axis activation and induction of corticotropin releasing factor expression (Lu et al, 2003;Liu et al, 2013).…”
Section: The Arcuate Nucleus Melanocortin Circuitrymentioning
confidence: 99%