2001
DOI: 10.1001/jama.286.17.2154
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Inflammatory Markers in Coronary Artery Disease

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Cited by 44 publications

(22 citation statements)
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“…This distinction between lesions versus lumen diameter challenges our traditional reliance upon coronary anatomy. As in the initiation and progression of arteriosclerosis, ample data support the involvement of inflammation in these thrombotic complications of arteriosclerosis [10][11][12][13] . The inflammatory stimuli produced by primary proinflammatory cytokines may undergo amplification through the induction of IL-6 production.…”
Section: Discussion
mentioning
confidence: 99%
How this paper cites the one you are viewing
“…This distinction between lesions versus lumen diameter challenges our traditional reliance upon coronary anatomy. As in the initiation and progression of arteriosclerosis, ample data support the involvement of inflammation in these thrombotic complications of arteriosclerosis [10][11][12][13] . The inflammatory stimuli produced by primary proinflammatory cytokines may undergo amplification through the induction of IL-6 production.…”
Section: Discussion
mentioning
confidence: 99%
How this paper cites the one you are viewing
“…Several mediators of this process, with varying biological roles, indicate the intensity of local and systemic inflammatory activity. The importance of these biomarkers is the potential to early identify patients who are vulnerable for developing cardiovascular events [17][18][19][20].…”
Section: Discussion
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confidence: 99%
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“…Inflammatory activity has been shown to predict myocardial infarction and stroke in patients with pre-existing cardiovascular disease (CVD). Numerous inflammatory parameters, such as cell adhesion molecules, cytokines, chemokines, and acute phase reactants, have been identified as valuable risk markers for the prediction of cardiovascular events (1,2). The inflammatory phase protein, C-reactive protein (CRP), is associated with an increased risk for CVD and stroke.…”
Section: Discussion
mentioning
confidence: 99%