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Cited by 24 publications
(23 citation statements)
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References 28 publications
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“…Further studies of remodeling processes in reperfused infarction in the CB2 −/− mice again confirmed the cardioprotective role of this receptor subtype [54]. In contrast to a rapid formation of granulation tissue and a compacted non-transmural scar in wild-type mice after 7 days of reperfusion, CB2 −/− mice showed a non-compacted transmural scar and a significantly worse cardiac function.…”
Section: Cb2 Receptor Knockout Mice (Cb2 −/− )mentioning
confidence: 60%
See 2 more Smart Citations
“…Further studies of remodeling processes in reperfused infarction in the CB2 −/− mice again confirmed the cardioprotective role of this receptor subtype [54]. In contrast to a rapid formation of granulation tissue and a compacted non-transmural scar in wild-type mice after 7 days of reperfusion, CB2 −/− mice showed a non-compacted transmural scar and a significantly worse cardiac function.…”
Section: Cb2 Receptor Knockout Mice (Cb2 −/− )mentioning
confidence: 60%
“…To begin with, circulating levels of endocannabinoids were elevated after cardiac injury, such as myocardial infarction in the rat [27] and ischemia/reperfusion in mice [28]. The CB2 receptors were upregulated in ischemic cardiomyocytes [28] and in cardiomyocytes following cultivation under hypoxia [29].…”
Section: Endocannabinoids and Cannabinoid Receptors In The Heartmentioning
confidence: 99%
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“…The MIinduced changes in the expression of endocannabinoid 2-AG synthesizing and metabolizing enzymes were comparable in both WT and CB2-/-mice. Given that global CB2 deficiency per se has multiple effects on cardiac healing responses, [6][7][8][9] we subsequently focused only on WT mice to study the consequences of pharmacological MAGL inhibition on acute post-MI inflammation and healing.…”
Section: Discussionmentioning
confidence: 99%
“…5 CB2 deficiency in mice (CB2-/-) leads to exaggerated myocardial ischemia-reperfusion injury and unfavorable post-myocardial infarction (MI) healing. [6][7][8][9] The beneficial effects of CB2 signaling in this setting involve cardioprotective mechanisms as well as anti-fibrotic and anti-inflammatory effects. 6,7,10 CB2 is most abundantly expressed on immune cells, including myeloid cells.…”
Section: Introductionmentioning
confidence: 99%