Horner's Syndrome in a Red-bellied Parrot (Poicephalus rufiventris)

Suspected cane toad ( Rhinella marina ) toxicity in a wedge‐tailed eagle ( Aquila audax)

“…The wedge‐tailed eagle presented with miosis rather than mydriasis, with the latter being an expected response to catecholamine derivatives in all domestic species. Birds exhibit conscious control of pupillary action due to the dominance of striated muscle fibres in the avian dilator muscle and show miosis variably during organophosphate poisoning 11 . Miosis can also be observed in birds following elevated intracranial pressure and acute uveitis 12,13 .…”

“…Birds exhibit  of  conscious control of pupillary action due to the dominance of striated muscle fibres in the avian dilator muscle and show miosis variably during organophosphate poisoning. 11 Miosis can also be observed in birds following elevated intracranial pressure and acute uveitis. 12,13 Malicious and incidental organophosphate toxicity is common in carrion-feeding raptors; however, the bird in this case was captive, lacked a known route of exposure, and displayed neurologic signs that did not respond swiftly to atropine administration.…”

Suspected cane toad ( Rhinella marina ) toxicity in a wedge‐tailed eagle ( Aquila audax)

“…The wedge‐tailed eagle presented with miosis rather than mydriasis, with the latter being an expected response to catecholamine derivatives in all domestic species. Birds exhibit conscious control of pupillary action due to the dominance of striated muscle fibres in the avian dilator muscle and show miosis variably during organophosphate poisoning 11 . Miosis can also be observed in birds following elevated intracranial pressure and acute uveitis 12,13 .…”

“…Birds exhibit  of  conscious control of pupillary action due to the dominance of striated muscle fibres in the avian dilator muscle and show miosis variably during organophosphate poisoning. 11 Miosis can also be observed in birds following elevated intracranial pressure and acute uveitis. 12,13 Malicious and incidental organophosphate toxicity is common in carrion-feeding raptors; however, the bird in this case was captive, lacked a known route of exposure, and displayed neurologic signs that did not respond swiftly to atropine administration.…”

Retrospective study of ocular disorders in Amazon parrots

“…In other species of parrots, some of reported ophthalmological disorders are: exophthalmos secondary to squamous cell carcinoma at the infraorbital sinus in a Solomon Island eclectus parrot (Eclectus roratus solomonensis) (Diaz-Figueroa et al 2006) and secondary to a retrobulbar adenoma in an African grey parrot (Psittacus erithacus) (Simova-Curd et al 2009); periorbital liposarcoma was also described in this species (Psittacus erithacus) (Graham et al 2003); Horner's syndrome secondary to trauma in a Redbellied parrot (Poicephalus rufiventris) (Gancz et al 2005), and mycobacterial keratitis in a Maximilian's parrot (Pionus maximiliani maximiliani) (Stanz et al 1995).…”

Determination of Reference Values for Tear Production and Intraocular Pressure in Pygoscelid Penguins of the Antarctic Peninsula † ‡

“…Normal STT-1 values [2,6,8,12,35,38,39] and IOP values measured with the Tonovet® rebound tonometer have been reported for various domestic and wild animals [1,2,5,9,12,14,24,30,35,[40][41][42]. However, very limited information is available on these ocular parameters in penguins living in their natural Antarctic habitat.…”

Suspected cane toad ( Rhinella marina ) toxicity in a wedge‐tailed eagle ( Aquila audax)

“…The wedge‐tailed eagle presented with miosis rather than mydriasis, with the latter being an expected response to catecholamine derivatives in all domestic species. Birds exhibit conscious control of pupillary action due to the dominance of striated muscle fibres in the avian dilator muscle and show miosis variably during organophosphate poisoning 11 . Miosis can also be observed in birds following elevated intracranial pressure and acute uveitis 12,13 .…”

“…Birds exhibit  of  conscious control of pupillary action due to the dominance of striated muscle fibres in the avian dilator muscle and show miosis variably during organophosphate poisoning. 11 Miosis can also be observed in birds following elevated intracranial pressure and acute uveitis. 12,13 Malicious and incidental organophosphate toxicity is common in carrion-feeding raptors; however, the bird in this case was captive, lacked a known route of exposure, and displayed neurologic signs that did not respond swiftly to atropine administration.…”

Retrospective study of ocular disorders in Amazon parrots

“…In other species of parrots, some of reported ophthalmological disorders are: exophthalmos secondary to squamous cell carcinoma at the infraorbital sinus in a Solomon Island eclectus parrot (Eclectus roratus solomonensis) (Diaz-Figueroa et al 2006) and secondary to a retrobulbar adenoma in an African grey parrot (Psittacus erithacus) (Simova-Curd et al 2009); periorbital liposarcoma was also described in this species (Psittacus erithacus) (Graham et al 2003); Horner's syndrome secondary to trauma in a Redbellied parrot (Poicephalus rufiventris) (Gancz et al 2005), and mycobacterial keratitis in a Maximilian's parrot (Pionus maximiliani maximiliani) (Stanz et al 1995).…”

Determination of Reference Values for Tear Production and Intraocular Pressure in Pygoscelid Penguins of the Antarctic Peninsula † ‡

“…Normal STT-1 values [2,6,8,12,35,38,39] and IOP values measured with the Tonovet® rebound tonometer have been reported for various domestic and wild animals [1,2,5,9,12,14,24,30,35,[40][41][42]. However, very limited information is available on these ocular parameters in penguins living in their natural Antarctic habitat.…”

Suspected cane toad ( Rhinella marina ) toxicity in a wedge‐tailed eagle ( Aquila audax)

“…The wedge‐tailed eagle presented with miosis rather than mydriasis, with the latter being an expected response to catecholamine derivatives in all domestic species. Birds exhibit conscious control of pupillary action due to the dominance of striated muscle fibres in the avian dilator muscle and show miosis variably during organophosphate poisoning 11 . Miosis can also be observed in birds following elevated intracranial pressure and acute uveitis 12,13 .…”

“…Birds exhibit  of  conscious control of pupillary action due to the dominance of striated muscle fibres in the avian dilator muscle and show miosis variably during organophosphate poisoning. 11 Miosis can also be observed in birds following elevated intracranial pressure and acute uveitis. 12,13 Malicious and incidental organophosphate toxicity is common in carrion-feeding raptors; however, the bird in this case was captive, lacked a known route of exposure, and displayed neurologic signs that did not respond swiftly to atropine administration.…”

Retrospective study of ocular disorders in Amazon parrots

“…In other species of parrots, some of reported ophthalmological disorders are: exophthalmos secondary to squamous cell carcinoma at the infraorbital sinus in a Solomon Island eclectus parrot (Eclectus roratus solomonensis) (Diaz-Figueroa et al 2006) and secondary to a retrobulbar adenoma in an African grey parrot (Psittacus erithacus) (Simova-Curd et al 2009); periorbital liposarcoma was also described in this species (Psittacus erithacus) (Graham et al 2003); Horner's syndrome secondary to trauma in a Redbellied parrot (Poicephalus rufiventris) (Gancz et al 2005), and mycobacterial keratitis in a Maximilian's parrot (Pionus maximiliani maximiliani) (Stanz et al 1995).…”

Determination of Reference Values for Tear Production and Intraocular Pressure in Pygoscelid Penguins of the Antarctic Peninsula † ‡

“…Normal STT-1 values [2,6,8,12,35,38,39] and IOP values measured with the Tonovet® rebound tonometer have been reported for various domestic and wild animals [1,2,5,9,12,14,24,30,35,[40][41][42]. However, very limited information is available on these ocular parameters in penguins living in their natural Antarctic habitat.…”