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“…Interestingly, disease association with HLA class I haplotypes is restricted to the HLA-B27 haplotype associated with ankylosing spondylitis, reactive arthritis, psoriatic arthritis, arthritis associated with inflammatory bowel disease, Reiter´s syndrome [4] and to HLA-A2 associated with Hashimoto's thyroiditis [5]. Disease association with HLA class II haplotypes is widespread, but DR2, DR3 and DR4 are prevailing haplotypes associated with the clinically more important diseases such as multiple sclerosis, systemic lupus erythematosus (SLE), insulin dependent diabetes mellitus (IDDM), and rheumatoid arthritis (RA) (surveyed in [5] and here updated).…”
Section: Association Of Hla and Autoimmunitymentioning
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“…Interestingly, disease association with HLA class I haplotypes is restricted to the HLA-B27 haplotype associated with ankylosing spondylitis, reactive arthritis, psoriatic arthritis, arthritis associated with inflammatory bowel disease, Reiter´s syndrome [4] and to HLA-A2 associated with Hashimoto's thyroiditis [5]. Disease association with HLA class II haplotypes is widespread, but DR2, DR3 and DR4 are prevailing haplotypes associated with the clinically more important diseases such as multiple sclerosis, systemic lupus erythematosus (SLE), insulin dependent diabetes mellitus (IDDM), and rheumatoid arthritis (RA) (surveyed in [5] and here updated).…”
Section: Association Of Hla and Autoimmunitymentioning
“…The disease association of HLA-B27 has also been interpreted in terms of epitope selection [4,10]. In addition, HLA-B27 β-chains have been shown to form homodimers that in turn are suspected of enhancing presentation of the disease inducing epitope, possibly to CD4+ T helper cells [11].…”
Section: Epitope Selection Vs Differential Expressionmentioning
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“…3,4 The strength of disease association with the HLA-B27 antigen varies markedly among the various SNSP forms as well as among different ethnic populations. 5 It was reported that in a study on 119 patients with HLA-B27-positive oligoarthritis during a follow-up period of 2-6 years, 25% progressed to a definite SNSA, 26% had recurrent oligoarthritis, and 34% became asymptomatic. 6 The combination of spondylitis, sacroiliitis, enthesopathy, arthritis of the extremities, and negative rheumatoid factor (RF) is common in the disease states of SNSA.…”
Section: Introductionmentioning
“…[2][3][4] These inflammatory reactions occur in individuals with a genetic predisposition initiated by bacterial antigens that interact with HLA-B27. 5 Sixty to 80% of patients with reactive arthritis are HLA-B27epositive. HLA-B27 functions to present endogenous peptides derived from intracellular proteins to the T-cell receptor on cytotoxic (CD8+) T lymphocytes.…”
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