1974
DOI: 10.1055/s-0028-1107950
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Grundlagen zur Bewertung leistungsphysiologischer Anpassungsvorgänge

Abstract: Grundlagen zur Bewertung leistungsphysiologischer AnpassungsvorgängeDie ständige Steigerung der körperlichen Belastung im Leistungssport wirft immer wieder die Frage auf, durch welche Anpassungsvorgänge die Höchstleistungen vollbracht werden und inwieweit Schädigungen möglich sind. In einem zusammenfassenden Oberblick sollen Anpassungsrnöglichkeiten des kardio-pulmonalen Systems und metabolische Veränderungen bei schwerer körperlicher Belastung besprochen werden. Die Kenntnis der physiologischen Anpassungsersc… Show more

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Cited by 31 publications
(7 citation statements)
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“…A stroke volume larger than normal has also been found by other investigators, who used invasive techniques (5,10,42,62). However, objections were made against such invasive results since they could hardly be gained under real resting conditions.…”
Section: Ah's Function At Restmentioning
confidence: 71%
“…A stroke volume larger than normal has also been found by other investigators, who used invasive techniques (5,10,42,62). However, objections were made against such invasive results since they could hardly be gained under real resting conditions.…”
Section: Ah's Function At Restmentioning
confidence: 71%
“…The sympathetic system is, as a result of its cardiocirculatory and metabolic (7) effects, important for an adequate substrate supply to the working musculature. Intensive endurance training leads to an increase of the stroke volume and of the maximum cardiac output, which leads to an improvement of the oxygen intake capability (4,15,16,26). The training-dependent adaptation of the autonomous nervous system, i.e., an increased vagotonic tone and a reduced sympathetic activity, is regarded to be the cause of the training bradycardia (5).…”
Section: Discussionmentioning
confidence: 99%
“…10) (31). During physical exercise the increase in contractility of the athlete's heart is reduced too, but this can not be seen as an indication of myocardial insufficency, but rather as the result of the decreased external sympathetic drive, since contractility reduction occurs without an increase in the enddiastolic pressure (15,16,28,31).…”
Section: MLmentioning
confidence: 99%