2008
DOI: 10.1590/s0100-736x2008001200007
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Gastric ulcers in pigs affected with postweaning multisystemic wasting syndrome

Abstract: 601Pesq. Vet. Bras. 28(12):601-605, dezembro 2008 RESUMO.-[Úlceras gástricas em suínos afetados pela síndrome multissistêmica do definhamento.] Amostras de linfonodos gástricos e os estômagos de 24 leitões selecionados de rebanhos afetados pela síndrome multissistê-mica do definhamento suíno e mortes súbitas por úlceras gástricas foram estudados. Os animais foram selecionados por baixa performance, baixo consumo de alimento e desnutrição. Os estômagos foram abertos, invertidos e classificados, conforme a se… Show more

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Cited by 7 publications
(3 citation statements)
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“…However, the ulcerations and erosions were more pronounced in the glandular mucosa than in pars oesophagea. The development of these lesions in glandular mucosa might be attributed to infectious diseases such as hog cholera, salmonellosis and PCV2 infections as well as dietary hygienic practices that disrupted the normal glandular mucosal epithelium (Correa et al, 2008;Omotosho et al, 2016). Another notable lesion in stomach was diphtheritic membrane formation in the glandular mucosa which was higher among growers.…”
Section: Resultsmentioning
confidence: 99%
“…However, the ulcerations and erosions were more pronounced in the glandular mucosa than in pars oesophagea. The development of these lesions in glandular mucosa might be attributed to infectious diseases such as hog cholera, salmonellosis and PCV2 infections as well as dietary hygienic practices that disrupted the normal glandular mucosal epithelium (Correa et al, 2008;Omotosho et al, 2016). Another notable lesion in stomach was diphtheritic membrane formation in the glandular mucosa which was higher among growers.…”
Section: Resultsmentioning
confidence: 99%
“…Meanwhile, pigs from the 10th percentile tADG had greater (P < 0.05) stomach inflammation incidence and severity compared to those from the 30th or 70th percentiles. Again, this effect again seems biologically irrelevant; it follows the same pattern of PCV2 incidence at 22 wk postweaning, and the pathogen has been clearly linked to lesions within the stomach and gastrointestinal tract (Chianini et al, 2003;Segalés et al, 2004;Corrêa et al, 2008) Another method to determine severity of pathogen load is to determine the concentration of markers of immunological or oxidative stress. An immune response results in the production of proinflammatory cytokines, the production of which requires energy and nutrients that may have otherwise been used for growth (Broussard et al, 2003;Johnson, 1997;Spurlock, 1997).…”
Section: Resultsmentioning
confidence: 99%
“…Meanwhile, Pigs from the 10 th percentile tADG had greater (P < 0.05) stomach inflammation incidence and severity compared to those from the 30 th or 70 th percentiles. Again, this effect again seems biologically irrelevant, it follows the same pattern of PCV2 incidence at 22-weeks post-weaning, and the pathogen has been clearly linked to lesions within the stomach and gastrointestinal tract (Chianini et al, 2003;Segalés et al, 2004;Corrêa et al, 2008) Another method to determine severity of pathogen load is to determine the concentration of immune markers, such as immunoglobulins, cytokines, chemokines, and glutathione. An immune response results in the production of pro-inflammatory cytokines, the production of which require energy and nutrients that may have otherwise been used for growth (Broussard et al, 2003;.…”
Section: Resultsmentioning
confidence: 99%