Frontal Lobe Syndrome in a Patient With Bilateral Globus Pallidus Lesions

Basal Ganglia Lesions and the Theory of Fronto-Subcortical Loops: Neuropsychological Findings in Two Patients with Left Caudate Lesions

“……”

Basal Ganglia Lesions and the Theory of Fronto-Subcortical Loops: Neuropsychological Findings in Two Patients with Left Caudate Lesions

“……”

Executive cognitive deficits in primary dystonia

“…Indeed in a meta‐analysis of 240 patients with basal ganglia lesions and movement disorders, 36% were found to have dystonic symptoms, with the putamen and globus pallidus the most frequently affected sites in these cases 39. A range of cognitive and behavioural deficits were also seen in this series, and there have been a number of other reports of either naturally occurring, or surgical lesions of the basal ganglia being associated with cognitive and behavioural sequelae, particularly of the dysexecutive type 15, 16, 39–42. As far as possible, the series of 14 patients reported here excluded any potential cases of secondary dystonia, where ed‐shift failure might be explained by the index brain insult.…”

Bilateral pallidal lesions following major haemorrhage: description of a case

“…To our knowledge, 10 cases have been reported in the literature of bilateral pallidal damage following global hypoxia [2][3][4]. Nine of them had prominent apathy and only one (case 5 from Laplane and col- [2]) had features resembling the symptoms of anxiety and disinhibition of our patient (anxiety and "lack of inhibition of motor programs").…”

Basal Ganglia Lesions and the Theory of Fronto-Subcortical Loops: Neuropsychological Findings in Two Patients with Left Caudate Lesions

“……”

Executive cognitive deficits in primary dystonia

“…Indeed in a meta‐analysis of 240 patients with basal ganglia lesions and movement disorders, 36% were found to have dystonic symptoms, with the putamen and globus pallidus the most frequently affected sites in these cases 39. A range of cognitive and behavioural deficits were also seen in this series, and there have been a number of other reports of either naturally occurring, or surgical lesions of the basal ganglia being associated with cognitive and behavioural sequelae, particularly of the dysexecutive type 15, 16, 39–42. As far as possible, the series of 14 patients reported here excluded any potential cases of secondary dystonia, where ed‐shift failure might be explained by the index brain insult.…”

Bilateral pallidal lesions following major haemorrhage: description of a case

“…To our knowledge, 10 cases have been reported in the literature of bilateral pallidal damage following global hypoxia [2][3][4]. Nine of them had prominent apathy and only one (case 5 from Laplane and col- [2]) had features resembling the symptoms of anxiety and disinhibition of our patient (anxiety and "lack of inhibition of motor programs").…”

Basal Ganglia Lesions and the Theory of Fronto-Subcortical Loops: Neuropsychological Findings in Two Patients with Left Caudate Lesions

“……”

Executive cognitive deficits in primary dystonia

“…Indeed in a meta‐analysis of 240 patients with basal ganglia lesions and movement disorders, 36% were found to have dystonic symptoms, with the putamen and globus pallidus the most frequently affected sites in these cases 39. A range of cognitive and behavioural deficits were also seen in this series, and there have been a number of other reports of either naturally occurring, or surgical lesions of the basal ganglia being associated with cognitive and behavioural sequelae, particularly of the dysexecutive type 15, 16, 39–42. As far as possible, the series of 14 patients reported here excluded any potential cases of secondary dystonia, where ed‐shift failure might be explained by the index brain insult.…”

Bilateral pallidal lesions following major haemorrhage: description of a case

“…To our knowledge, 10 cases have been reported in the literature of bilateral pallidal damage following global hypoxia [2][3][4]. Nine of them had prominent apathy and only one (case 5 from Laplane and col- [2]) had features resembling the symptoms of anxiety and disinhibition of our patient (anxiety and "lack of inhibition of motor programs").…”