Poster Presentations 2017
DOI: 10.1136/annrheumdis-2017-eular.2872 View full text |Buy / Rent full text
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Abstract: BackgroundAPS pathophysiology is not clear enough yet since it has been implicated that aPL can activate cells (endothelial cells, monocytes, platelets), interfere with hemostatic reactions and activate complement reactions [1,2].ObjectivesThe aim of this study was to evaluate oxidative stress markers and it relations to endothelial damage as risk factor for thrombosis in patients with primary (PAPS) and secondary (SAPS) antiphospholipid syndrome (APS) in correlation to traditional risk factors.MethodsFlow med… Show more

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