1956
DOI: 10.1001/archderm.1956.01550070065012
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Fox - Fordyce Disease

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Cited by 15 publications
(6 citation statements)
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“…Our study revealed several new findings, such as perifollicular mucin, adventitial fibrosis, and increased mast cell density, that can be used as clues to the diagnosis of FFD. These features have been mentioned in past works, 5,10 but curiously these are not mentioned in standard textbook descriptions of FFD.…”
Section: Commentmentioning
confidence: 99%
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“…Our study revealed several new findings, such as perifollicular mucin, adventitial fibrosis, and increased mast cell density, that can be used as clues to the diagnosis of FFD. These features have been mentioned in past works, 5,10 but curiously these are not mentioned in standard textbook descriptions of FFD.…”
Section: Commentmentioning
confidence: 99%
“…2 The so-called sweat retention vesicle has been reputed to be the singular diagnostic feature. 3 Although the clinical features of FFD are relatively characteristic, 4,5 rendering a specific histopathologic diagnosis is less straightforward. The search for a retention vesicle via conventional serial sections is often frustrating if not futile, although transverse sectioning can be used to demonstrate a vesicle.…”
mentioning
confidence: 99%
“…Consistent with the idea that FFD represents a disorder of infundibular keratinization and subsequent apocrine duct obstruction, the disease, and specifically, the pruritus, is often exacerbated in summer months or with exercise or sweating. [1][2][3][4][5] Nonablative laser therapy has emerged as a useful approach for selective hair removal. [6][7][8] In the procedure, discrete wavelengths of light target melanin pigment in the hair bulb and shaft.…”
Section: Ox-fordyce Disease (Ffd)mentioning
confidence: 99%
“…1,[3][4][5][9][10][11][12][13][14][15][16] There is general agreement that dilation of the follicular infundibulum with hyperkeratosis and keratin plugging is a common feature of FFD. 1,4,5,[9][10][11][12][13]15,16 Keratin plugging may be caused by keratinocyte dysmaturation, manifested by dyskeratosis and parakeratosis-the latter often resembling a cornoid lamella. 1,5,10,15 Blockage of the apocrine duct at the point of entry to the epidermis obstructs apocrine secretion 4 and causes apocrine duct and gland dilation.…”
Section: Commentmentioning
confidence: 99%
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