Focal myositis associated with S‐1 radiculopathy: Report of two cases

Streichenberger, Nathalie; Meyronet, David; Fière, Vincent; Pellissier, Jean‐François; Petiot, P.

doi:10.1002/mus.10540

Cited by 30 publications

(9 citation statements)

References 17 publications

(8 reference statements)

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“…Treatment is focused on rhabdomyolysis which require adequate hydration to reduce serum myoglobin and protection of renal function. In all of these conditions, counseling is essential to limit drug abuse (Streichenberger et al, 2004; Chawla et al, 2005; O’Connor and McMahon, 2008; Brancaccio et al, 2010; Kasaoka et al, 2010). …”

Section: Step 1: Overview and Understanding Of Myopathies In Systemicmentioning

confidence: 99%

Stepwise Approach to Myopathy in Systemic Disease

Chawla¹

2011

Front. Neur.

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Muscle diseases can constitute a large variety of both acquired and hereditary disorders. Myopathies in systemic disease results from several different disease processes including endocrine, inflammatory, paraneoplastic, infectious, drug- and toxin-induced, critical illness myopathy, metabolic, and myopathies with other systemic disorders. Patients with systemic myopathies often present acutely or sub acutely. On the other hand, familial myopathies or dystrophies generally present in a chronic fashion with exceptions of metabolic myopathies where symptoms on occasion can be precipitated acutely. Most of the inflammatory myopathies can have a chance association with malignant lesions; the incidence appears to be specifically increased only in patients with dermatomyositis. In dealing with myopathies associated with systemic illnesses, the focus will be on the acquired causes. Management is beyond the scope of this chapter. Prognosis is based upon the underlying cause and, most of the time, carries a good prognosis. In order to approach a patient with suspected myopathy from systemic disease, a stepwise approach is utilized.

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Section: Step 1: Overview and Understanding Of Myopathies In Systemicmentioning

confidence: 99%

Stepwise Approach to Myopathy in Systemic Disease

Chawla¹

2011

Front. Neur.

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“…However, as an uncommon finding, particularly in the case of chronic partial denervation, an enlargement of the partially denervated muscles may occur 6. This may be caused by pseudohypertrophy with replacement of muscle fibers by fatty tissue, or by edema, focal myositis,8 or true hypertrophy induced by spontaneous repetitive motor unit discharges. Because there were no signal changes on unenhanced T1‐weighted images in our patient, we consider fatty replacement unlikely.…”

Section: Discussionmentioning

confidence: 99%

Denervation hypertrophy may mimic local tumor spread on magnetic resonance imaging

et al. 2006

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We report a patient with an extensive paranasal sinus carcinoma. One year after tumor resection, magnetic resonance imaging (MRI) showed swelling of the ipsilateral masticatory muscles with signal increase on T2-weighted images and gadolinium-DTPA uptake, suggestive of local tumor infiltration. However, electromyography, biopsy, and follow-up MRI confirmed denervation pseudohypertrophy of the muscles innervated by the mandibular nerve and excluded tumor recurrence. Muscle denervation and pseudohypertrophy should be considered in the differential diagnosis of appropriate patients with suspected tumor recurrence.

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“…En revanche, les observations suivantes de myosites sont possiblement reliées à la myosite focale d'Heffner : myosites du mollet après cure chirurgicale pour sciatique radiculaire S1 chez deux patients [18] ; myosite focale après prise de venlafaxine, avec disparition chez ce patient des signes dix jours après l'arrêt du médicament incriminé, sans essai de réintroduction [19] ; myosite focale des jambes après une vaccination par le BCG [20] ; myosite focale avec atteinte des deux mollets chez un patient hémodialysé [21]. L'association myosite focale/thyroïdite a été signalée et invite à étudier les liens éventuels avec des maladies systémiques.…”

Section: Les Confins De La Myosite Focaleunclassified