volume 8, issue 10, Pe78244 2013
DOI: 10.1371/journal.pone.0078244
View full text
|
Sign up to set email alerts
|
Share

Abstract: The microvascular endothelium of the kidney glomerulus is injured in Shiga-like toxigenic bacterial infection, genetic or acquired loss of complement regulatory protein function, and allo-immune responses of solid-organ or bone marrow transplantation. Existing models of diseases with glomerular endothelial cell (EC) injury, collectively grouped as thrombotic microangiopathies, are problematic, impeding investigation of the mechanisms of microvascular defense and repair. To develop a model of glomerular endothe…

Expand abstract

Search citation statements

Order By: Relevance

Citation Types

1
4
0

Paper Sections

0
0
0
0
0

Publication Types

0
0
0
0

Relationship

0
0

Authors

Journals