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Cited by 43 publications
(45 citation statements)
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References 24 publications
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“…Gomes et al (2003) demonstrated that monocytes from IND produce more IL-10 compared to CARD, suggesting that these cells may play an important role in regulating immune response during chronic Chagas disease. On the other hand, CARD patients present a higher ability to produce TNF-α, suggesting a role for pro-inflammatory monocytes in the development of cardiac disease (Vitelli-Avelar et al 2008). These findings corroborate with previous reports analyzing isolated peripheral blood mononuclear cells (PBMC) (Correa-Oliveira et al 1999, Golgher & Gazzinelli 2004, Dutra et al 2005).…”
Section: Innate Immune Response During Late Chronic Chagas Diseasesupporting
confidence: 90%
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“…Gomes et al (2003) demonstrated that monocytes from IND produce more IL-10 compared to CARD, suggesting that these cells may play an important role in regulating immune response during chronic Chagas disease. On the other hand, CARD patients present a higher ability to produce TNF-α, suggesting a role for pro-inflammatory monocytes in the development of cardiac disease (Vitelli-Avelar et al 2008). These findings corroborate with previous reports analyzing isolated peripheral blood mononuclear cells (PBMC) (Correa-Oliveira et al 1999, Golgher & Gazzinelli 2004, Dutra et al 2005).…”
Section: Innate Immune Response During Late Chronic Chagas Diseasesupporting
confidence: 90%
“…The analyses of pro and anti-inflammatory cytokine profiles of circulating monocytes and NK cells have recently been reported (Gomes et al 2003, Vitelli-Avelar et al 2008. Gomes et al (2003) demonstrated that monocytes from IND produce more IL-10 compared to CARD, suggesting that these cells may play an important role in regulating immune response during chronic Chagas disease.…”
Section: Innate Immune Response During Late Chronic Chagas Diseasementioning
confidence: 99%
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“…During the initial phases of infection, macrophages produce cytokines such as interleukin 12 and TNF-α that induce production of interferon gamma (IFN-γ). This IFN-γ, produced by CD4 + and CD8 + T-lymphocytes, is essential for the control of infection (Chessler et al, 2009;Sathler-Avelar et al, 2008;Sathler-Avelar et al, 2009;Une et al, 2003). In macrophages activated by IFN-γ, intracellular parasite growth is controlled through nitric oxide (NO) production.…”
Section: Immunologic Mechanisms For Parasite Control and Heart Damagementioning
confidence: 99%
“…Whereas, Bustamante et al (Bustamante et al, 2008) showed that when Bz is administered for 40 days, parasitological cure and stable and protective CD8+ T central memory response are obtained. Furthermore, Bz itself can modulate the immune response during indeterminate and chronic Chagas disease (Sathler-Avelar et al, 2008;SathlerAvelar et al, 2009). In addition, persistent parasitemia with T. cruzi, along with male gender, is an independent predictor of progression of Chagas cardiomyopathy in some studies .…”
Section: Role Of Anti-chagasic Therapy In the Chronic Cardiomiopathymentioning
confidence: 99%