1994
ETCO2 monitoring during low flow states: clinical aims and limits
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Cited by 46 publications
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Abstract
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“…As CO 2 is highly soluble and the gas exchange surface area is large in a mature lung, the primary factors regulating end-tidal CO 2 levels are the CO 2 partial pressure gradient between alveolar gas and blood and the rate of pulmonary perfusion. As such, endtidal and mixed venous CO 2 levels can be used to estimate pulmonary perfusion and cardiac output [14]. In the neonate immediately after birth, although both pulmonary perfusion and CO 2 partial pressure gradients likely influence ECO 2 levels once gas exchange commences, our data indicate that the degree of lung aeration at end inflation (when gas volumes and gas exchange surface area are maximal) is the predominant factor.…”
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confidence: 79%