Erratum: Corrigendum: B cell–helper neutrophils stimulate the diversification and production of immunoglobulin in the marginal zone of the spleen

Abstract

In the version of this article initially published, the second affiliation for Stephanie C. Ganal is missing. This author is also affiliated with the Spemann Graduate School of Biology and Medicine, Freiburg, Germany. The error has been corrected in the HTML and PDF versions of the article.Corrigendum: Signaling by Fyn-ADAP via the Carma1-Bcl-10-MAP3K7 signalosome exclusively regulates inflammatory cytokine production in NK cells In the version of this article initially published online, the affiliation of author Bart Vanhaesebroeck was incorrect. The correct affiliation is as follows: Center for Cell Signaling, Barts Cancer Institute, Queen Mary University of London, London, UK. The error has been corrected for the print, PDF and HTML versions of this article. Erratum: Matching cellular dimensions with molecular sizes Michael Reth Nat. Immunol. 14, 765-767 (2013)In the version of this article originally posted online, the units in the text referring to Figure 1a were incorrect. The correct unit is "μm" and the correct text is " the receptor depicted would measure about 3 μm, compared with a resting B lymphocyte, with an average diameter of about 7 μm. " The error has been corrected in this file as of 22 August 2013.Erratum: The role of the immune system in governing host-microbe interactions in the intestine In the version of this article initially published, a label was missing from Figure 2. The lymphoid structure in the large intestine should be labeled 'Isolated lymphoid follicle' . The error has been corrected in the HTML and PDF versions of the article.c o r r i g e n d a a n d e r r ata npg

Tumor suppressor maspin as a modulator of host immune response to cancer

“…In addition, we also demonstrated that the activation of neutrophils by maspin may be B-cell dependent since we showed that mice bearing maspin expressing tumors had elevated expression of tumor-cell reactive and maspin-specific immunoglobulin G (IgG). Although B-cell response is thought to be primarily dependent on CD4 + T-cell interaction, recent studies showed that neutrophils can promote the differentiation and activation of B-cells independently of CD4 + T-cells [37,38]. This was the first evidence demonstrating the function of maspin as an epithelial-specific antigen in cancer disease.…”