Epicardial adipose tissue thickness and type 2 diabetes risk according to the FINDRISC modified for Latin America

Lima-Martínez, Marcos M.; Colmenares, Leomar; Campanelli, Yanei; Paoli, Mariela; Rodney, Marianela; Santos, Raul D.; Iacobellis, Gianluca

doi:10.1016/j.artere.2018.06.004

Cited by 1 publication

(1 citation statement)

References 29 publications

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“…Moreover, it has been proposed that the adipokines released by EAT in pathological conditions have paracrine effects on cardiac electrical activity, affecting conductivity and promoting atrial fibrillation [ 20 ], and in coronary arteries, they cause atherosclerosis through the promotion of inflammation and immune cell infiltration [ 21 , 22 ]. Lately, EAT thickness has been considered a clinical biomarker that correlates to features of heart failure and metabolic syndrome [ 23 , 24 , 25 , 26 , 27 ]. Another fat depot that surrounds the coronary arteries is the perivascular adipose tissue (PVAT).…”

Section: Adipose Tissue Dysfunction and Cvdsmentioning

confidence: 99%

Adipokines and Inflammation: Focus on Cardiovascular Diseases

Feijóo‐Bandín

Aragón-Herrera

Moraña-Fernández

et al. 2020

IJMS

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It is well established that adipose tissue, apart from its energy storage function, acts as an endocrine organ that produces and secretes a number of bioactive substances, including hormones commonly known as adipokines. Obesity is a major risk factor for the development of cardiovascular diseases, mainly due to a low grade of inflammation and the excessive fat accumulation produced in this state. The adipose tissue dysfunction in obesity leads to an aberrant release of adipokines, some of them with direct cardiovascular and inflammatory regulatory functions. Inflammation is a common link between obesity and cardiovascular diseases, so this review will summarise the role of the main adipokines implicated in the regulation of the inflammatory processes occurring under the scenario of cardiovascular diseases.

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