1999
DOI: 10.1002/(sici)1099-0690(199910)1999:10<2533::aid-ejoc2533>3.0.co;2-o
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EPC Syntheses of Trifluorocitronellol and of Hexafluoropyrenophorin – A Comparison of Their Physiological Properties with the Nonfluorinated Analogs

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Cited by 26 publications

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“…In a second approach (cf. B in Scheme 8), the aldol addition of the oxazolidinone derivatives III with aldehydes IV (PG' Tr [107]; PG' Ts [106]), followed by deoxygenation under Barton ± McCombie conditions [67] [108] [ 109], resulted in the isolation of degradation products caused by retro-aldol reactions, occurring during the deoxygenation step. Thus, the amidomethylation reaction via Ti-enolates was attempted (cf.…”
mentioning
confidence: 99%
Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task.
“…In a second approach (cf. B in Scheme 8), the aldol addition of the oxazolidinone derivatives III with aldehydes IV (PG' Tr [107]; PG' Ts [106]), followed by deoxygenation under Barton ± McCombie conditions [67] [108] [ 109], resulted in the isolation of degradation products caused by retro-aldol reactions, occurring during the deoxygenation step. Thus, the amidomethylation reaction via Ti-enolates was attempted (cf.…”
mentioning
confidence: 99%
Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task.
Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task.
Exaggerated anticipatory anxiety is common in social anxiety disorder (SAD). Neuroimaging studies have revealed altered neural activity in response to social stimuli in SAD, but fewer studies have examined neural activity during anticipation of feared social stimuli in SAD. The current study examined the time course and magnitude of activity in threat processing brain regions during speech anticipation in socially anxious individuals and healthy controls (HC). Method Participants (SAD n = 58; HC n = 16) underwent functional magnetic resonance imaging (fMRI) during which they completed a 90s control anticipation task and 90s speech anticipation task.