Endovascular Management of Arteriovenous Malformations of the Brain

Bruno, Charles; Meyers, Philip M.

doi:10.1159/000346927

Cited by 51 publications

(50 citation statements)

References 149 publications

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“…AVM is a typical lesion of vascular malformations. The prevalence of brain AVM is <1% and its incidence is between 0.01 and 0.001% . The incidence of AVM per 100 000 person‐years is 1.1–1.4 .…”

Section: Discussionmentioning

confidence: 99%

Giant intracranial arteriovenous malformation as the focus of epileptic seizures

et al. 2017

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A man in his late thirties was found in a supine position in the hallway of his house. He had been diagnosed with epilepsy at approximately 20 years old. Since stopping treatment, epileptic events occurred more frequently and his condition deteriorated in the past 2 years. Autopsy revealed that head injuries were found on the left side of his head. A fracture from the left parietal bone to the anterior cranial fossa was also detected. A subdural hemorrhage (hematoma) spanned a wide range. A subarachnoid hemorrhage was also identified in the left parietal region. His brain weighed 1603 g, was edematous, and showed right uncal herniation. In the right cerebral hemisphere, a thick, enlarged blood vessel ran from the sagittal sinus. An egg-sized tumorous lesion of blood vessels was found on the bottom of the frontal lobe. This vascular lesion had formed between the sagittal sinus and right anterior cerebral artery. Pathologically, veins and arteries were found together, and, thus, this case was diagnosed as an arteriovenous malformation (AVM). No other pathological and toxicological findings were observed. Subdural hematoma, the cause of death, occurred from the fall to the floor. An epileptic seizure may have been the cause of the fall. AVM on his brain was considered to be the focal lesion of epileptic seizures.

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Section: Discussionmentioning

confidence: 99%

Giant intracranial arteriovenous malformation as the focus of epileptic seizures

et al. 2017

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“…AVM is a typical lesion of vascular malformation. The prevalence of brain AVM is <1 % and its incidence is between 0.01 and 0.001 % . The incidence of AVM per 100 000 person‐years is 1.1–1.4 .…”

Section: Discussionmentioning

confidence: 99%

“…The prevalence of brain AVM is <1 % and its incidence is between 0.01 and 0.001 %. 5 The incidence of AVM per 100 000 person-years is 1.1-1.4. 6-10 Seventeen percent of patients presented during the first two decades of life, while 20 % presented in the seventh and eighth decades.…”

Section: Discussionmentioning

confidence: 99%

Basal subarachnoid hemorrhage by rupture of arteriovenous malformation at the cerebellopontine angle

et al. 2017

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A man in his late forties had lived as a recluse for more than ten years. He was found dead in his room. At autopsy, subarachnoid hemorrhage (SAH) was detected at the base of the brain, which weighed 1333 g. The cerebellar tonsil was swollen. The cerebral ventricle was enlarged and filled with blood. A hematoma was observed in the upper part of the left side of the cerebellar hemisphere. The location and size of SAH in this case indicated that the rupture of a cerebral aneurysm (CA) had occurred; however, CA was not detected. A mass of blood vessels buried in the hematoma was observed at the left cerebellopontine angle (CPA). The vascular lesion showed round-shaped blood vessels as well as flat-shaped vessels with the appearance of veins, but with elastic fibers indicative of arteries. The lesion was considered to be the nidus and was 5-8 mm in size. Feeding arteries appeared to be from the anterior inferior cerebellar artery (AICA). However, the draining vein and anastomotic parts of the artery and vein were not confirmed. Based on these histopathological features, this vascular lesion was diagnosed as arteriovenous malformation (AVM). A differential diagnosis between AVM at CPA and CA is needed in order to identify the source of non-traumatic SAH.

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“…To achieve cure, the anatomic exclusion of the nidus is required because partial embolization does not afford protection from hemorrhage and triggers over time recruitment of new vasculature to the nidus. 5,18 The primary endovascular approach for AVMs is traditionally performed by selective arterial catheterization of the nidus. 5,6,19 Consequently, the feasibility of arterial access is the key aspect to successful treatments using conventional endovascular methods.…”

Section: Endovascular Therapy In Childrenmentioning

confidence: 99%