2012
DOI: 10.1177/030089161209800421 View full text |Buy / Rent full text
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Abstract: EML4-ALK-positive lung cancer is a novel cancer entity associated with light or never smoking, younger age, and adenocarcinoma with acinar or signet-ring cell type histology. Another mutation of ALK with NPM, resulting in NPM-ALK fusion mutation, was described in patients with anaplastic large cell lymphoma (ALCL). It was subsequently reported in organ transplant recipients and patiens undergoing immunosuppressive therapy. We describe a case of lung cancer in a 36-year-old nonsmoking woman with ulcerative coli… Show more

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“…Crizotinib was administered after unsuccessful chemotherapy. The authors report having obtained a response with a prolonged treatment but a reduction in the drug dosage was necessary due to diarrhea [59].…”
Section: Digestive System Diseasesmentioning
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“…Crizotinib was administered after unsuccessful chemotherapy. The authors report having obtained a response with a prolonged treatment but a reduction in the drug dosage was necessary due to diarrhea [59].…”
Section: Digestive System Diseasesmentioning
“…This finding of nervous system-related drugs, including selective serotonin reuptake inhibitors, being potentially suitable for treating lung cancer is not an independent event, as aducanumab, a drug for Alzheimer's disease (Schneider, 2020), has also been Frontiers in Pharmacology frontiersin.org experimentally tested for lung cancer and encouraging results were observed (Morrison, 2016). Azathioprine has previously been reported to be used for treating patients with LUAD, but its effect and mechanism of action are unknown (McAdam et al, 1974;Lazarev et al, 2012;Baik et al, 2018). Ethanol has the potential to suppress CL1-5 human LUAD cell migration, and it inhibits matrix metalloproteinase-2/9 via the extracellular signal-regulated kinase, c-Jun N-terminal kinase, p38, and phosphatidylinositol 3-kinase/Akt signalling pathways (Chen et al, 2012).…”
Section: Discussionmentioning
“…Azathioprine has previously been reported to be used for treating patients with LUAD, but its effect and mechanism of action are unknown ( McAdam et al, 1974 ; Lazarev et al, 2012 ; Baik et al, 2018 ). Ethanol has the potential to suppress CL1-5 human LUAD cell migration, and it inhibits matrix metalloproteinase-2/9 via the extracellular signal-regulated kinase, c-Jun N-terminal kinase, p38, and phosphatidylinositol 3-kinase/Akt signalling pathways ( Chen et al, 2012 ).…”
Section: Discussionmentioning